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NSG 6005 FINAL EXAM STUDY GUIDE / NSG6005 FINAL EXAM STUDY GUIDE (LATEST, 2020): SOUTH UNIVERSITY | UPGRADED AND PERFECT GUIDE, DOWNLOAD TO SCORE A| $15.49   Add to cart

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NSG 6005 FINAL EXAM STUDY GUIDE / NSG6005 FINAL EXAM STUDY GUIDE (LATEST, 2020): SOUTH UNIVERSITY | UPGRADED AND PERFECT GUIDE, DOWNLOAD TO SCORE A|

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NSG 6005 FINAL EXAM STUDY GUIDE / NSG6005 FINAL EXAM STUDY GUIDE (LATEST, 2020): SOUTH UNIVERSITY | UPGRADED AND PERFECT GUIDE, DOWNLOAD TO SCORE A|

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  • August 26, 2020
  • 48
  • 2020/2021
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By: STERLINGBAS • 3 year ago

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NSG 6005 FINAL STUDY GUIDE


Guanfacine- used to treat HTN or ADHD, non stimulant. Alpha 2 adrenergic agonist.
Antiarrhythmics start on p. 322
Class I: sodium channel blockers
Class Ia: lengthens action potential (quinidine, procainamide[requires freq.dosing],
disopyramide)
Class Ib: shortens action potential (lidocaine, phenytoin) treats ventricular arrhythmias
Class Ic: minimal or no effect on action potential; severe ventricular tachycardias- where
no other drugs have worked
Class II: beta blockers (propranolol, metoprolol, atenolol)- indirectly reduces slope by blocking
chronotropic impact of norepinephrine
Atenolol has longer half-life than metoprolol
Class III: agents that lengthen action potential (potassium channel blockers)–(amiodarone,
bretyllium) effective in treating re-entry problems, inhibits v-fib due to myocardial ischemia,
improves contractility
Class IV: Calcium channel blockers(verapamil, diltiazem, bepridil)& (amlodipine, felodipine)
2 types of CCB:
type I- non-hydropyridines-affect conduction through AV node, have neg chronotropic effect
(verapamil, diltiazem)
type II:hydorphyridines-do not affect conduction through AV node (nifedipine, amlodipine,
felodipine)
Class IA & IC -what is similar?
Lidocaine-
Amiodaraone effective against supraventricular rhythms


BPH & HTN med
Doxazosin- used to treat BPH & HTN
Mexiletine- only available orally

, Valerian has no adverse reactions when used at the recommended level; however, overdosage at
2.5 g or more can cause cardiac disturbance, excitability, headache, insomnia, and nausea. It can
potentiate alcohol and other CNS depressants if taken in large amounts. (p. 139)


Ayurvedic medicine- Although all three doshas exist together, often plants and people are
classified by the one that is most dominant in them, referred to as the person’s “Prakruti,” and
specific to them as an individual. (p. 132)
Glaucoma meds:
Longer eyelashes-lanasoprost or bimatoprost – prostaglandin drugs
Glaucoma med cause blurriness after instilled into eyes for few minutes
Procainamide-short acting; need dose reduction in CHF and renal impairment (p. 325)
VLDL- synthesized in the liver (p. 1130) Drugs that inhibit VLDL synthesis in the liver (niacin,
fibric acid derivatives) also reduce LDLs via the endogenous pathway (p.1130).


Cholesterol meds:
-acted on sterols?
Inhibitor Hmgcoa- statins
Bile from liver-bile sequestrins
Cost benefit analysis
Cardiotonic
Digoxin with renal failure- contradicted in renal impairment; Digoxin can also be problematic
when treating older adults and patients with renal insufficiency. Renal function may decrease
during heart failure treatment and the drug may not be adequately excreted, allowing it to
increase to toxic levels. Digoxin levels should be closely monitored in these patients. (p. 1072)
Because digoxin is excreted essentially unchanged by the kidneys, severe renal impairment
effectively contraindicates its use. (p. 318)


Treatment with hepatitis C- antiviral meds, NS5A inhibitors
Tacrine-
Timolol-

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