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NR507 Advanced Pathophysiology NR 507 Midterm Adv Patho Latest Exam Questions with Correct Answers | 100% Pass Guaranteed | Graded A+ | $14.99   Add to cart

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NR507 Advanced Pathophysiology NR 507 Midterm Adv Patho Latest Exam Questions with Correct Answers | 100% Pass Guaranteed | Graded A+ |

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NR507 Advanced Pathophysiology NR 507 Midterm Adv Patho Latest Exam Questions with Correct Answers | 100% Pass Guaranteed | Graded A+ |

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  • November 21, 2024
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  • 2024/2025
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Chamberlain University
NR507 Advanced Pathophysiology
NR 507 Midterm and Final Exam

Course Title and Number: NR507 Advanced Pathophysiology
Exam
Exam Title: NR 507 Exam
Exam Date: Exam 2024- 2025
Instructor: [Insert Instructor’s Name]
Student Name: [Insert Student’s Name]
Student ID: [Insert Student ID]

Examination
180 minutes
Instructions:
1. Read each question carefully.
2. Answer all questions.
3. Use the provided answer sheet to mark your responses.
4. Ensure all answers are final before submitting the exam.
5. Please answer each question below and click Submit when you have completed
the Exam.
6. This test has a time limit, The test will save and submit automatically when the
time expires
7. This is Exam which will assess your knowledge on the course Learning
Resources.


Good Luck!

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NR507 Advanced Pathophysiology 2024-2025
NR 507 Midterm Adv Patho Latest Exam Questions with
Correct Answers | 100% Pass Guaranteed | Graded A+ |
Read All Instructions Carefully and Answer All the
Questions Correctly Good Luck: -

Hypertension - =Answer>> consistent elevation of
systemic arterial blood pressure, increases in cardiac
output or total peripheral resistance or both. CO is
increased by any condition that increases HR or SV,
whereas peripheral resistance is increased by any
factor that increases blood viscosity or reduces vessel
diameter (vasoconstriction)

primary hypertension - =Answer>> Essential or
idiopathic hypertension
Genetic and environmental factors
Affects 92% to 95% of individuals with hypertension

secondary hypertension - =Answer>> high blood
pressure caused by the effects of another disease.
Example (renal artery stenosis, kidney atrophy,
increase in plasma renin)

calcium binding - =Answer>> Binding of calcium to
subunit of troponin
Induces conformation change in troponin
Troponin-tropomyosin complex moved
Myosin binding sites of actin exposed

Troponin - =Answer>> regulatory protein that binds
to actin, tropomyosin, and calcium. Integral to muscle
contraction in skeletal muscle and cardiac muscle, but
not smooth muscle



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Cardiac Output (CO) - =Answer>> volume of blood
ejected by each ventricle per minute
CO= HRx SV

factors that affect cardiac output - =Answer>>
preload
after-load
heart rate
myocardial contractility

Ejection Fraction (EF) - =Answer>> calculation of how
much blood a ventricle can eject with one contraction
SV / EDV
55-70% of ventricular blood ejected/ systole

cardiac contractility (inotropic) - =Answer>> Force
generated at any given end diastolic volume
Increased:
sympathetic stimulation (anxiety, increased thyroxine)

Decreased:
low ATP levels (inschemia, hypoxia, acidosis)

preload (venous return) - =Answer>> volume of blood
in ventricles at end of diastole

Increased: CHF, hypervolemia
Decreased: cardiac tamponade, hypovolemia

AFTERLOAD (arteries) - =Answer>> resistance to left
ventricular ejection (systole)

Increased: COPD, hypertension, valve dx (all r/t pulm
htn)

Decreased: hypotension, vasodilation (shock)


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