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MSN 570 ADVANCE PATHO MAIN TEST WITH QUESTIONS AND ANSWERS
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MSN 570 ADVANCE PATHO MAIN TEST WITH QUESTIONS AND ANSWERS
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MSN 570 ADVANCE PATHO MAINTEST WITH QUESTIONS AND
ANSWERS
MI h- hcorrect hanswer-Death hof hmyocardium hfrom hsudden hblockage hof hcoronary
hartery hblood hflow
What hcauses hcell hdeath hin hMI? h- hcorrect hanswer-unstable hplaque hbreaks hoff,
htriggering hplatelet haggregation, hcoagulation hcascade hand hthrombus hformation.
hcontinued hno hblood hflow hin h10 hseconds hischemia, h20 hminutes hcell hdie
What hconsequence hof hcell hdeath hleads hto hCP? h- hcorrect hanswer-hypoxia has
hcannot hmeet hcell hdemand hfor hoxygen
How his hMI hdiagnosed h- hcorrect hanswer-EKG h(STEMI) hor helevated hTroponin
h(non-STEMI)
What hmight hyou hfind hin hblood hconsistent hwith hMI? h- hcorrect hanswer-elevated
hCK hMB, hTroponin
Where hdoes hCK hMB hand hTroponin hcome hfrom? h- hcorrect hanswer-Release hof
hlysosomal henzymes; hCK hMB hdetected hafter hmyocardial hnecrosis, hTroponin
hreleased hfrom hdamaged hheart hmuscle
3 hmechanism hof hcell hinjury hrelated hto hreperfussion hinjury h- hcorrect hanswer-
ischemia, hnecrosis, hreperfussion hl/t hRAS
, define hreperfussion hinjury h- hcorrect hanswer-injury hthat hoccurs hafter hoxygen his
hreintroduced hto htissue hperfusion
how hdoes hinflammatory hprocess hcontribute hto hreperfussion hinjury? h- hcorrect
hanswer-as hoxygen his hreintroduced hit hcomes hin hcontact hwith hdamaged hproteins
htriggering hinflammatory hresponse
how hdoes hoxidative hprocess hcontribute hto hRI? h- hcorrect hanswer-RAS hcauses
himpairment hof hcell hmembrane hand hpro-apotptoic hcells hrelease. hmaking hcell
hunstable
what his hprimary hHypertension? h- hcorrect hanswer-Essential hor hidiopathic,
helevated hBP hwithout han hidentified hcause, haccounts hfor h90 h- h95% hof hall hcases
hof hhypertension?
what his hsecondary hhypertension? h- hcorrect hanswer-Elevated hBP, hlabile hBP;
hrelated hto hconditions hie hkidney hdz; hresistant hto hBP hmeds
what his hsignificance hof hVentricular hhypertrophy hand hHTN? h- hcorrect hanswer-
persistent hhypertension hleads hto hventricular hhypertrophy; hdecrease hCO
what hcomorbidities/conditions hdoes hHTN hcause? h- hcorrect hanswer-stroke,
hischemia, hheart hfailure, haneurysm, hhemorrhage
what his hpreload? h- hcorrect hanswer-volume hof hblood hin hventricles hat hend hof
hdiastole
what his hafterload? h- hcorrect hanswer-peripheral hresistance hagainst hwhich hthe hleft
hventricle hmust hpump
hoe hdoes hHTN hl/t hCHF? h- hcorrect hanswer-overwork hleft hventricle hdue hto
hincrease hpressure; hdecreased hpumping hability hof hheart hand hdecreases hCO.
identify hpathological hprocess hof hgangrene? h- hcorrect hanswer-form hof hcoagulative
hnecrosis; hinterruption hof hblood hflow hl/t hdenaturing hcells henzymes hand hbacterial
hinvasion
three htypes hof hgangrene h- hcorrect hanswer-dry, hwet h& hgas
what his hthe hmajor htreatment happroach hfor hgangrene? h- hcorrect hanswer-
removing haffective htissue hto hprevent hinfection; htreating hproblem hthat hled hto
hgangrene
why huse hantibiotic hfor hgangrene? h- hcorrect hanswer-to hslow hbacterial hdamage
What hare hthe htwo hmajor hnecrotic hprocesses? h- hcorrect hanswer-Coagulative hand
hliquifactive
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