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NURS 5315 ADV PATHO EXAM 1 QUESTIONS AND ANSWERS

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  • NURS 5315 Advanced Pathophysiology
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  • NURS 5315 Advanced Pathophysiology

NURS 5315 ADV PATHO EXAM 1 QUESTIONS AND ANSWERS

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  • September 24, 2024
  • 445
  • 2024/2025
  • Exam (elaborations)
  • Questions & answers
  • nurs 5315
  • NURS 5315 Advanced Pathophysiology
  • NURS 5315 Advanced Pathophysiology
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470 Multiple choice questions

Definition 1 of 470
What happens once the antigen-antibody complex is formed in Type III hypersensitivities?

The complex causes the body to produce more antibodies

Once the complex is formed, they are deposited in vessel walls or extravascular tissue
and trigger an immune and inflammatory response


The complex enhances the immune response without inflammation

The complex is immediately eliminated by the body

Definition 2 of 470
treatment for PCP pneumocystis Jiroveci pneumonia

Sarco-

Bactrim

AB positive

Edema

Definition 3 of 470
coagulative, liquefactive, caseous, fat, gangrenous

etiology of edema

interstitial pattern of fluid shift


metabolic acidosis

5 Types of Necrosis

,Definition 4 of 470
pH >7.45
Paco2 normal
Bicarb >26

T:


Ck enzymes

Kinin system Activated


Metabolic alk
Uncompensated

Definition 5 of 470
What does alloimmunity imply?

Describes a benign reaction to environmental factors


Indicates the body's failure to produce any antibodies

Refers to the immune system attacking its own cells

Implies the formation of antibodies to foreign antigens (exogenous antigens)

Definition 6 of 470
E-byproduct of purine degradation
CI-accumulation gets deposited into the tissues of the kidney, heart, earlobes, joints
-liver can produce more uric acid than can be eliminated
-diuretics can trigger kidneys to increase absorption of uric acid
-over accumulation or under secretion of uric acid can result in gout

Urate accumulation Effects and Clinical Implications

Metastatic Calcification Effects and Clinical Implications

Acid Base Balance effect on K+

Hepatocellular damage:Alcoholic Hepatitis

,Definition 7 of 470
from GI losses or diuretics
C.M. Volume depletion, orthostatic hypotension, tachycardia, lack of organ perfusion

hypermagnesemia


hyperkalemia

hypovolemic hypernatremia

Hypovolemic Hypernatremia etilolgy

Definition 8 of 470
seizures, coma, areflexia, incontinence, death

Tumor Marker:Beta Human Chorionic gonadotropin

Tumor Marker:Alpha Fetoprotein

AB


Serum Na level: < 120mEq/L clinical manifestations

Definition 9 of 470
E. increased acid production, loss of bicarb, diminished renal excretion of hydrogen
C.M hyperventilation (compensatory), h/a, n/v/d, dehydration, hypotension
pH <7.4 HCO3 <22

metabolic acidosis

Dystrophic Calcification

Oncotic pressure

Hypercalcemia

, Definition 10 of 470
hydrostatic pressure promotes the movement of about 10% of fluid along with small amount of
protein into lymphatics which eventually returns to the circulation

Clinical manifestations of fluid volume deficit

Growth factors

Interstitial pattern of fluid shift

Fluid volume deficit pathophysiology

Definition 11 of 470
Type III chronic, multi-system, inflammatory disease, Primarily in women 20-40
Antibodies formed against DNA and nucleus
Butterfly rash, photosensitivity

Systemic Lupus Erythematosus


Coagulation cascade activated

Type III Hypersensitivity

Euvolemic Hypernatremia

Definition 12 of 470
What does IgE do upon initial exposure to the allergen?

Igg and c3b may also bind to the antigen and the macrophages and trigger cell lysis
through phagocytosis

It is a t-cell mediated reaction that occurs 11-14 days after first exposure and 5-6 days
after the second exposure. this results in mononuclear infiltration, decreased circulation
and tissue necrosis.

It binds with the allergen


It primes the stage for a reaction to occur later

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