Exam (elaborations)
NURS 5315 GI MODULE 9, EXAM 5 QUESTIONS AND ANSWERS
- Course
- Institution
NURS 5315 GI MODULE 9, EXAM 5 QUESTIONS AND ANSWERS
[Show more]
Content preview
Name: Score:68 Multiple choice questions
Definition 1 of 68
Normally a transient condition d/t persistent or rising bilirubin that manifests during the first
week of life in healthy, full term infants and subsides after a couple of weeks. Caused by mild
unconjugated hyperbilirubinemia.
Gilbert's syndrome
Peptic ulcer disease
Infant cretinism
Physiologic neonatal jaundice
Definition 2 of 68
May not cause any S&S if they are small. Some can pass into the common bile duct and cause
an obstruction which will cause painful spasms and contraction of the bile duct in the RUQ
area called biliary colic. Pain is also felt in the back, right should or right scapula. N/V.
Small Bowel Obstruction
Crohn's Disease S&s
Alcoholic Cirrhosis
Gallstones S&S
Definition 3 of 68
Abnormally high blood pressure in the portal venous system caused by resistance to portal
blood flow. Commonly caused by fibrosis, obstruction from cirrhosis, thrombosis, or narrowing
of hepatic portal vein. Most common S&S is vomiting blood from bleeding esophageal varices.
Acute Pancreatitis
Ascites
Portal Hypertension
Jaundice
,Definition 4 of 68
Hepatitis B surface antigen +
Hepatitis B core antibody IgM +
Hepatitis B Surface Antibody -
means what
Acute Hepatitis B Infection
Chronic Hepatitis B infection
Acute Pancreatitis Diagnosis And Complications
Chronic hepatitis C infection
Definition 5 of 68
Caused by the presence of a nonabsorbable substance in the intestines. This pulls water by
osmosis into the intestinal lumen and results in large volume diarrhea. This is how mag citrate,
lactulose and miralax work. Causes include: excessive ingestion of nonabsorbable sugars, tube
feedings, dumping syndrome, malabsorption, pancreatic enzyme deficiency, bile salt
deficiency, small intestine bacterial overgrowth or celiac disease
Motility diarrhea
Ascites
Osmotic diarrhea
Pyloric stenosis
,Definition 6 of 68
Onset is 24 hours after birth; often d/t hemolytic disease of the newborn; seen in exclusive
breastfed infants, premature infants, ABO or RH incompatibility, maternal age greater than 25,
male infant, delayed meconium passage and birth trauma.
bilirubin levels >20 or indirect bilirubin >15.
Patho: increased bilirubin production, impaired hepatic uptake or excretion of unconjugated
bilirubin, or a delayed maturation of liver conjugating mechanisms
Pancreatic cancer
Motility diarrhea
Immune mediated hemolysis
Pathologic neonatal jaundice
Definition 7 of 68
Incidence increases with age and is more common in males and african americans. Risk factors:
ETOH use, family history, smoking, non O blood type, DM type 2, and chronic pancreatitis. K-
ras mutation, a proto-oncogene is most common genetic alteration.
Tumors arise from exocrine cells of pancreas ducts, called adenocarcinomas; tumors of the
head of pancreas grow quickly and can obstruct the portal veins and common bile duct
Ascites
Colon cancer
Motility diarrhea
Pancreatic cancer
, Definition 8 of 68
mouth,pharynx, esophagus, stomach, and duodenum
Upper GI organs
Upper Gi bleed
Lower Gi bleed
Cholelithiasis
Definition 9 of 68
inflammation of the gallbladder from obstruction in biliary tract. S&S: precipitated by a fatty
meal, GERD, positive murphy's sign and rebound tenderness. Lab abnormalities include
leukocytosis, increased alkaline phosphatase and direct bilirubin.
Appendicitis
Cholecystitis
Jaundice
Acute Pancreatitis
Definition 10 of 68
Characterized as the presence of diverticula in the large intestine. Risk factors include older
age, genetic predisposition, obesity, smoking, diet, lack of exercise, ASA and other NSAIDS,
altered DI microbiome and abnormal colonic peristalsis
Colon cancer
Appendicitis
Crohn's disease
Diverticular disease
The benefits of buying summaries with Stuvia:
Guaranteed quality through customer reviews
Stuvia customers have reviewed more than 700,000 summaries. This how you know that you are buying the best documents.
Quick and easy check-out
You can quickly pay through credit card or Stuvia-credit for the summaries. There is no membership needed.
Focus on what matters
Your fellow students write the study notes themselves, which is why the documents are always reliable and up-to-date. This ensures you quickly get to the core!
Frequently asked questions
What do I get when I buy this document?
You get a PDF, available immediately after your purchase. The purchased document is accessible anytime, anywhere and indefinitely through your profile.
Satisfaction guarantee: how does it work?
Our satisfaction guarantee ensures that you always find a study document that suits you well. You fill out a form, and our customer service team takes care of the rest.
Who am I buying these notes from?
Stuvia is a marketplace, so you are not buying this document from us, but from seller selftest. Stuvia facilitates payment to the seller.
Will I be stuck with a subscription?
No, you only buy these notes for $9.00. You're not tied to anything after your purchase.
Can Stuvia be trusted?
4.6 stars on Google & Trustpilot (+1000 reviews)
72042 documents were sold in the last 30 days
Founded in 2010, the go-to place to buy study notes for 14 years now