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NUR 3125 - Exam #2 Latest Update Actual Exam 150 Questions and 100% Verified Correct Answers Guaranteed A+ Approved by the Professor $20.49   Add to cart

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NUR 3125 - Exam #2 Latest Update Actual Exam 150 Questions and 100% Verified Correct Answers Guaranteed A+ Approved by the Professor

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NUR 3125 - Exam #2 Latest Update Actual Exam 150 Questions and 100% Verified Correct Answers Guaranteed A+ Approved by the Professor

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  • September 23, 2024
  • 18
  • 2024/2025
  • Exam (elaborations)
  • Questions & answers
  • NUR 3125
  • NUR 3125
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NUR 3125 - Exam #2 Latest Update 2024-2025
Actual Exam 150 Questions and 100% Verified
Correct Answers Guaranteed A+ Approved by
the Professor
_____ and ______ indicates infection in urine test - CORRECT ANSWER: nitrite &
leukocyte esterase


______ will compensate first, _______ will lag behind - CORRECT ANSWER: lungs,
kidneys


3 theories of nephrolithiasis - CORRECT ANSWER: 1. supersaturation of urine by stone
forming constituents
2. deposition of calcium phosphate -> plaque becomes a calculi
3. deficiency of one or all proteins that inhibit stone formation


acute coronary syndrome (ACS) - CORRECT ANSWER: unstable angina to MI (cardiac
emergency)


acute hypercapnic respiratory failure - CORRECT ANSWER: unable to maintain a level
of alveolar ventilation sufficient to eliminate CO2 and keep arterial O2 levels within
normal range - ventilatory FAILURE


acute hypoxemic respiratory failure - CORRECT ANSWER: mismatch of ventilation and
perfusion (lungs ventilated but not perfused) - NOT oxygenating


acute kidney injury - CORRECT ANSWER: decreased GFR, increased BUN &
creatinine


ARDS - CORRECT ANSWER: diffuse alveolar injury, pulmonary capillary damage,
bilateral pulmonary infiltrates and severe hypoxemia
- severity evaluated by PF ratio

,asthma - CORRECT ANSWER: hyper reactive airway disease, chronic inflammatory
disease, bronchial hyper reactivity, REVERSIBLE AIRWAY CONSTRICTION


patho:
- presence of EOSINOPHILS, lymphocytes, mast cells
- IgE mediated
- TNF-a, IL-4 and IL-5
- eosinophils release LEUKOTRIENES -> release of HISTAMINE from mast cells


clinical man:
WHEEZING, cough, dyspnea, chest tightness, USE OF ACCESSORY MUSCLES
(abdominal/intercostal)


atelectasis - CORRECT ANSWER: incomplete expansion of a lung or portion of lung


atypical pneumonia - CORRECT ANSWER: caused viral and mycoplasma infections
that involve alveolar septum and interstitium of lung (patchy)
- m. pneumoniae
- influenza
- RSV


less striking symptoms


BUN - CORRECT ANSWER: increases azotemia when:
1. kidney is dysfunctional
2. HIGH protein diet is consumed
3. DEHYDRATION concentrates blood urea

, calcium oxalate - CORRECT ANSWER: 75%
- hyperabsorption


chronic bronchitis - CORRECT ANSWER: - hypersecretion of mucus and edema inhibit
ventilation
- cannot get air IN
- cough for 3 MONTHS OUT OF THE YEAR FOR 2 CONSECUTIVE YEARS
- chronic hypoxia
- clubbing of fingernails
- blue boater


chronic ischemic heart disease - CORRECT ANSWER: recurrent, transient myocardial
ischemia and stable angina


chronic renal failure - CORRECT ANSWER: - usually progresses to ESRD
- hemodialysis or kidney transplant are the ONLY options to support life


clinical manifestations of acute kidney injury - CORRECT ANSWER: METABOLIC
ACIDOSIS, volume overload -> pulmonary edema, encephalopathy, HYPERKALEMIA,
CVA tenderness, HEMATURIA, PROTEINURIA, OLIGURIA (< 400 mL urine output per
day or < 20 mL per hour)


clinical manifestations of ARDS - CORRECT ANSWER: rapid onset (12-18 hrs),
tachycardia, marked hypoxemia (refractory hypoxemia), BILATERAL INFILTRATES,
non-compliant lung (very stiff)


clinical manifestations of fluid volume deficit - CORRECT ANSWER: dark urine, DRY
mucous membranes, low urine output, orthostatic hypotension, poor skin turgor


clinical manifestations of fluid volume excess - CORRECT ANSWER: ascites, crackles
in lungs, dyspnea, edema, weight gain (2 # = 1 L fluid)

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