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CARDIOLOGY BOARDS ABIM EXAM 2 LATEST VERSIONS ACTUAL EXAM 250 QUESTIONS AND CORRECT DETAILED ANSWERS WITH RATIONALES $12.49   Add to cart

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CARDIOLOGY BOARDS ABIM EXAM 2 LATEST VERSIONS ACTUAL EXAM 250 QUESTIONS AND CORRECT DETAILED ANSWERS WITH RATIONALES

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CARDIOLOGY BOARDS ABIM EXAM 2 LATEST VERSIONS ACTUAL EXAM 250 QUESTIONS AND CORRECT DETAILED ANSWERS WITH RATIONALES What is a positive stress test - Answer️️ -Flat or Down sloping St-segment depression >1 mm occurring 80 msec after j point When to stop a stress test - Answer️️ -St...

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  • September 18, 2024
  • 34
  • 2024/2025
  • Exam (elaborations)
  • Questions & answers
  • CARDIOLOGY BOARDS ABIM
  • CARDIOLOGY BOARDS ABIM
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EmillyCharlotte
TITLE: EMILLYCHARLOTTE 2024/2025 ACADEMIC PERIOD
OWNER: EMILLYCHARLOTTE
COPYRIGHT STATEMENT: ©2024 EMILLYCHARLOTTE. ALL RIGHTS RESERVED
FIRST PUBLISHED: SEPTEMBER 2024

CARDIOLOGY BOARDS ABIM EXAM 2 LATEST VERSIONS
2024-2025 ACTUAL EXAM 250 QUESTIONS AND CORRECT
DETAILED ANSWERS WITH RATIONALES


What is a positive stress test - Answer✔️✔️-Flat or Down sloping St-segment depression

>1 mm occurring 80 msec after j point


When to stop a stress test - Answer✔️✔️-St segment depression > 2 mm, ventricular

tachycardia, drop in SBP > 15, chest pain, dyspnea, lightheadedness

Stress test of choice with a LBBB or ventricular pacing? - Answer✔️✔️-Myocardial

perfusion imaging with adenosine,NOT exercising!

Know the algorithm for stress testing - Answer✔️✔️-See page 5-3,figure 5-1

When to not use doutamine for stress - Answer✔️✔️-History of VT, severe HTN, Low BP,

poor echo images

When to not use adenosine for stress - Answer✔️✔️-Bronchospasm, severe valvular

dysfunction, severe carotid stenosis, 2nd degree heart block, theophylline dependent

Normals for PA catheter pressures - Answer✔️✔️-RA <7, RV 30/7, PCWP 3-11

PA cath findings in tamponade or restrictive pericarditis - Answer✔️✔️-Diastolic

pressures elevated and equalized in all chambers, low BP

PA cath findings with RV AMI - Answer✔️✔️-Elevated RA and PA pressures, decreased

or nl PCWP, hypotension, and inferior MI. R side is decompensated, cannot fill L side of

the heart

,TITLE: EMILLYCHARLOTTE 2024/2025 ACADEMIC PERIOD
OWNER: EMILLYCHARLOTTE
COPYRIGHT STATEMENT: ©2024 EMILLYCHARLOTTE. ALL RIGHTS RESERVED
FIRST PUBLISHED: SEPTEMBER 2024

PA cath findings in cardiogenic shock - Answer✔️✔️-Elevated PCWP, RA pressure, and

decreased SBP/cardiac output

PA cath findings in mitral stenosis with RV failure - Answer✔️✔️-Elevated RA, PA (very

elevated), PCWP, nl SBP

PA cath findings in pulmonary HTN - Answer✔️✔️-Elevated PA, RA pressures, nl PCWP,

SBP

Pulsus paradoxus - Answer✔️✔️-decrease in systolic BP of more than 10mmHg with

normal inspiration; palpated as weakened pulse with inspiration along with more heart

contractions to pulse beats

What conditions give you pulsus paradoxus? - Answer✔️✔️-Constrictive or restrictive

pericarditis, asthma, tension pneumothorax

What gives you pulsus bisferiens (two systolic peaks per cycle) - Answer✔️✔️-Aortic

regurgitation, HOCM

What causes pulsus alternans - Answer✔️✔️-Severe LV dysfunction

What causes pulsus tardus - Answer✔️✔️-Aortic stenosis

How do positional maneuvers affect blood flow and murmurs - Answer✔️✔️--

standing/valsalva - decreased cardiac filling, decreases most murmurs except MVP and

HOCM

-squatting/ lying down - increase cardiac volume, increased murmurs except MVP,

HOCM

,TITLE: EMILLYCHARLOTTE 2024/2025 ACADEMIC PERIOD
OWNER: EMILLYCHARLOTTE
COPYRIGHT STATEMENT: ©2024 EMILLYCHARLOTTE. ALL RIGHTS RESERVED
FIRST PUBLISHED: SEPTEMBER 2024
-sustained handgrip - increases systemic resistance, decreases murmur in HOCM, AS

What causes a physiologic split S2 - Answer✔️✔️-Increased blood volume in the RV

prolongs systole and delays pulmonary valve closure

What causes a fixed split S2 - Answer✔️✔️-Pulmonary stenosis, PE, LV pacer, RBBB,

MR (early AV closure), ASD, RV failue

What causes a paradoxic split S2 - Answer✔️✔️-LBBB, RV pacing, HOCM

What causes an S3? - Answer✔️✔️-Rapid LV filling - acute ventricular decompensation,

severe AR or MR

KNOW - S3 with LV dysfunction is a poor prognostic factor - Answer✔️✔️-...

What causes a S4? - Answer✔️✔️-Decreased ventricular compliance during atrial

contraction - ischemic heart dz, AS, MR, HOCM, hypertrophic or diabetic

cardiomyopathy, HTN heart dz, concentric LVH

Can you have a S4 with atrial fibrillation? - Answer✔️✔️-No - no atrial contraction

What are the parts of the venous waveform? - Answer✔️✔️-A wave - atrial contraction

X descent - atria relax, RV fills rapidly

Bottom of x descent is TC valve closure

V wave - ventricle contacting against closed TC valve

Y descent - TC valve opens, passive emptying into ventricle

What gives elevated a and v waves - Answer✔️✔️-Pulmonary HTN, RV infarction

Large r side v waves - Answer✔️✔️-Septal rupture

, TITLE: EMILLYCHARLOTTE 2024/2025 ACADEMIC PERIOD
OWNER: EMILLYCHARLOTTE
COPYRIGHT STATEMENT: ©2024 EMILLYCHARLOTTE. ALL RIGHTS RESERVED
FIRST PUBLISHED: SEPTEMBER 2024

Large v waves - Answer✔️✔️-TR (right), MR (left)

Rapid x and y descent - Answer✔️✔️-Constrictive pericarditis, restrictive cardiomyopathy,

tamponade (x descent only, loss of y descent)

Large a waves - Answer✔️✔️-TS,severe RVH (on right), MS

Cannon a waves - Answer✔️✔️-AV disassociation - complete heart block, ventricular

pacing

Slow Y descent - Answer✔️✔️-Delayed atrial emptying - TS

Most important prognostic factor with CAD - Answer✔️✔️-Degree of LV dysfunction

Causes of resting ST elevation - Answer✔️✔️-MI, pericarditis, LV aneurysm, LBBB,

ventricular pacing, LVH, early repolarization

Hibernating myocardium - Answer✔️✔️-myocardium near the infarction may be

underperfused but not necrotic-the metabolism of the cells adapts to low energy

supplies and are nonfunctional until perfusion is restored

Reperfusion injury - Answer✔️✔️-the re-establishment of blood flow after a coronary

artery is blocked, which may further damage the heart tissue due to the formation of

oxygen free radicals

Stunned myocardium - Answer✔️✔️-prolonged post ischemic dysfunction, salvaged by

reperfusion, several days

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