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NUR 3125 – INFECTION FINAL EXAM REAL EXAM 100+ QUESTIONS AND CORRECT ANSWERS (VERIFIED ANSWERS) A GRADE $12.99   Add to cart

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NUR 3125 – INFECTION FINAL EXAM REAL EXAM 100+ QUESTIONS AND CORRECT ANSWERS (VERIFIED ANSWERS) A GRADE

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NUR 3125 – INFECTION FINAL EXAM REAL EXAM 100+ QUESTIONS AND CORRECT ANSWERS (VERIFIED ANSWERS) A GRADE blocks immune response (bacterial resistance) - Answer-release surface molecules that bind to and neutralize antibodies fungi structures - Answer-mold, yeast, or dimorphic mycosis - A...

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  • September 17, 2024
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  • 2024/2025
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  • nur 3125 infection
  • NUR 3125 – INFECTION
  • NUR 3125 – INFECTION
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NUR 3125 – INFECTION FINAL EXAM
REAL EXAM 100+ QUESTIONS AND
CORRECT ANSWERS (VERIFIED
ANSWERS) A GRADE

blocks immune response (bacterial resistance) - Answer-release surface molecules that
bind to and neutralize antibodies

fungi structures - Answer-mold, yeast, or dimorphic

mycosis - Answer-infection with a fungus

fungal infection transmission routes... - Answer-inhalation or contamination of wounds

dermatophytes - Answer-fungal infection of skin, hair, or nails

how does the fungus attach? - Answer-adhere to epithelial cells using several
polysaccharides on the fungal surface

do fungal infection pathogens produce toxins? what is the exception? - Answer-NO, but
Candida albicans is needed for the infection of mucosa

typical immune and inflammatory response to fungal infections... - Answer-cell-mediated
response with infiltration of T-cells, macrophages, and neutrophils

difficulties with dealing with fungal infections... - Answer-encapsulation and altered
antigen expression --> progressive production of pro-inflammatory cytokines -->
destruction of healthy tissue

fungal pathogens adapt to host environment

survive phagocytosis by replicating in the phagosome

protozoan infection examples - Answer-Toxoplasma gondii= causes blindness and
miscarriage

Trichomonas vaginalis= a sexually transmitted infection that triggers premature delivery

parasitic infection examples - Answer-intestinal and tissue nematodes (e.g., hookworm,
roundworm)

, flatworms (e.g., liver fluke, lung fluke, tapeworm)

main transmission for parasitic and protozoan infections... - Answer-vectors (ex. malaria
by mosquitos, trypanosomes by tsetse fly, Leishmania spp. by sand fleas)

extracellular parasites... - Answer-attach to a tissue lining, and evolve to a more
invasive form

intracellular parasites... - Answer-reproduce within cells

parasitic infection may insert pores into intestinal cells leading to... - Answer-increased
influx of calcium, efflux of water, and diarrhea

parasites can survive immune destruction by... - Answer-invade macrophages

block recognition of pathogen antigens by binding with antibodies

block the immune response by:
secreting enzymes that degrade immunoglobulins, inactivate the complement system,
prevent antigen processing by macrophages, inhibit complement-mediated phagocyte
chemotaxis, inhibit T-cell function

basic structure of a virus... - Answer-virion (nucleic acid surrounded by the capsid)

how is an infectious virus classified... - Answer-is it DNA or RNA?

does it use enzyme reverse transcriptase for replication?

what is the most common affliction of humans? - Answer-viral diseases (ex. common
cold, cold sores, hepatitis, HIV)

virus life cycle... - Answer-attaches to the host cell via protein receptors --> penetrates
the host cell --> releases genetic information into the host cytoplasm

interferons are effective against

RNA virus life cycle... - Answer-enter the host nucleus -->
produce messenger RNA (mRNA) (new viral material).
may produce provirus DNA (retroviruses, HIV)

DNA virus life cycle... - Answer-DNA viruses enter the host nucleus --> may integrate
into the host DNA; may make mRNA

harmful effects of viruses... - Answer-inhibition of DNA, RNA, or protein synthesis in
host cells

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