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Exam (elaborations)

NURS 351 Exam 1B

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NURS 351 Exam 1B NURS 351 Exam 1B NURS 351 Exam 1B

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  • September 15, 2024
  • 19
  • 2024/2025
  • Exam (elaborations)
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  • NURS 351
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lectjoseph
NURS 351 Exam 1B
pain is a - ANS protective mechanism



pain is the most common - ANS symptom for which people seek medical treatment



optimal pain management causes - ANS -quicker rate of recovery

-better functioning

-fewer postoperative complications



pain is the signal between - ANS periphery, spinal cord, brain



simple reflex arc - ANS response at spinal cord level, does not involve the brain



nociceptors - ANS pain nerves



spinal cord - ANS pain signals up to brain through ascending tract (spinothalamic tract)



brain sends - ANS outgoing signals to modify pain, endorphins



neurotransmitters - ANS -enhance/inhibit pain signals

-acetylcholine, GABA, serotonin, etc



simple reflex arc:



afferent neuron

interneuron

efferent neuron - ANS afferent neuron: incoming signal to the dorsal horn of the spinal cord



interneuron: connector

,efferent neuron: outgoing signal from the anterior spinal cord



a protective response that do not involve brain input



what interprets pain signals - ANS the brain



spinothalamic tract - ANS primary ascending tract for pain signals from spinal cord to brain



corticospinal tract - ANS outgoing signals from he brain travel on this descending tract



Nociception - ANS the response of the nervous system to painful stimuli



nociceptors are - ANS pain receptors



A-delta (10%) - ANS large, myelinated fibers that carry initial acute signal of pain (sharp, stinging,
cutting, pinching)



C-fibers (90%) - ANS Small, unmyelinated fibers that carry signals of dull, persistent pain (dull,
burning, aching)



neurotransmitters and pain, can be - ANS excitatory or inhibitory



what manipulation is used for pain management? - ANS pharmacological manipulation



neurochemicals & actions:



prostaglandins (from Cox 1 enzymatic pathway) - ANS enhances inflammation, pain, edema



neurochemicals & actions:



interleukins - ANS enhances inflammation, pain, edema

, neurochemicals & actions:



tumor necrosis factor - ANS enhances inflammation, edema, pain, decreases appetite



neurochemicals & actions:



leukotrienes - ANS enhances inflammation, edema, and bronchospasm, esp. in asthma and allergy



neurochemicals & actions:



bradykinins - ANS enhances inflammation



neurochemicals & actions:



glutamate - ANS amplifies pain signal



neurochemicals & actions:



Substance P - ANS amplifies pain signal



neurochemicals & actions:



enkephalins, endorphins - ANS inhibitory influence on pain; natural opioid



neurochemicals & actions:



acetylcholine - ANS inhibitory action on pain in the spinal cord



neurochemicals & actions:



gamma-aminobutyric acid - ANS inhibitory action on pain in the spinal cord AND brain

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