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SDSU Nurs 323 - Quiz 3 Questions and Correct Answers $8.99   Add to cart

Exam (elaborations)

SDSU Nurs 323 - Quiz 3 Questions and Correct Answers

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  • Course
  • NUR 323
  • Institution
  • NUR 323

What is coronary artery disease? heart disease caused by impaired coronary blood flow Where is the left main artery? main vessel that comes off left ventricle - supplies whole left side of heart "widow maker" What happens if left main artery is occluded? left side loses ability to contract Where...

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  • September 13, 2024
  • 18
  • 2024/2025
  • Exam (elaborations)
  • Questions & answers
  • NUR 323
  • NUR 323
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twishfrancis
SDSU Nurs 323 - Quiz 3 Questions and
Correct Answers
What is coronary artery disease? ✅heart disease caused by impaired coronary blood
flow

Where is the left main artery? ✅main vessel that comes off left ventricle - supplies
whole left side of heart
"widow maker"

What happens if left main artery is occluded? ✅left side loses ability to contract

Where is the left anterior descending artery? ✅from front and bottom of left ventricle

What happens if the left anterior descending artery is occluded? ✅bottom of ventricle
loses ability to contract

Where is the circumflex? ✅covers side and back of left ventricle - feeds left atrium

What does the right coronary artery feed? ✅right side of heart

What happens if the right coronary artery is occluded? ✅whole right side of heart loses
ability to contract

What is the pathogenesis of coronary artery disease? ✅endothelium of healthy arteries
autoregulates coronary blood flow - dilate when need for increase o2

Atherosclerosis causes a fail in... ✅autoregulation

Atherosclerosis causes a narrowing of ___________. Over time, ________ need will
exceed supply ✅vessels - o2

What is a stable plaque? ✅obstructs flow but does not travel - has thick fibrous cap

What is an unstable plaque? ✅can rupture causing platelets to stick - becomes
thrombus

What is included in plaque vulnerability? ✅thin fibrous cap w/ large lipid core
Hemodynamic triggers
Vessel tension
Sympathetic surge

,What are hemodynamic triggers? ✅blood flow turbulence - high turbulence causes
plaque to break off

What is vessel tension? ✅stretch of vessel wall - the more the stretch, the less likely it
will break off - the stiffer it is, the more likely it will break

What is sympathetic surge? ✅things that push hr and stress

What are examples of sympathetic surge? ✅emotions
Physical - cold, running, etc

Lipid core stimulates platelets to stick which stimulates.... ✅clotting cascade..thrombus
formed..decreased perfusion...ischemia and/or necrosis
Most commonly occurs in the 1st hour after waking

What are acute coronary syndromes? ✅spectrum of heart disease problems - starts w/
unstable angina to mi

Classifications of acute coronary syndromes are based on the presence or absence of
what? ✅ecg, serum biomarkers

What will an ecg show in an acute coronary syndrome? ✅st elevation or depressoin

What are serum biomarkers? ✅troponin i and t
Ck-mb (isoenzyme)

What is the most specific indicator of mi? ✅troponin i and t

When do troponin levels rise? ✅within 3 hours of damage
Can be measured for 7-10 days after mi

What is the normal troponin range? ✅0-0.05 ng/dl

What is ck-mb? ✅creatine kinase myocardial band

When does ck-mb elevate? ✅4-8 hours
Returns to normal within 2-3 days

What is the type of pain associated with an unstable angina/non-st elevation myocardial
infarction (nstemi)? ✅pain that persists and occurs at rest and last more than 20 mins
w/ no nitroglycerine given
Pain that persists and is severe w/ a new onset
More severe and prolonged pain that is increasing in frequency

, What causes nstemi? ✅unstable plaque that ruptures
Vessel obstruction that causes constriction or spasms within bv - leads to narrowing of
coronary lumen
Inflammation

Unstable angina will not have what? ✅elevated markers and st seg elevation

Nstemi -> infarction -> ✅damage to tissue that causes release of biomarkers
- main difference between unstable angina and mi
- ecg findings

What causes stemi? ✅atherosclerosis -> complete vessel occlusion -> lack of blood
flow to myocardium
O2 demand exceeds supply -> anaerobic metabolism -> build up of lactic acid
Compensatory mechanisms -> increase in hr -> vasoconstriction of peripheral vessels -
> causes more damage

What pain is associated w/ stemi? ✅pressure radiating from left shoulder/arm - may
also go up neck and into jaw

What pain might women feel w/ stemi? ✅atypical chest pain - shoulder hurts, pain in
elbow, nauseated

What gi symptoms might a pt feel with stemi? ✅nausea, heart burn/reflux
Treat gi symptoms to rule out

What skin changes happen w/ stemi? ✅pallor or flushed, diaphoretic and clammy

W/ a stemi, when does cardiac cell necrosis occur? ✅20-40 mins biomarkers are
released

What are pathologic changes associated w/ mi? ✅transmural injury
Subendocardial injury

What is a transmural injury? ✅goes full thickness of heart wall
That part is no longer able to contract/pump

What happens to st seg w/ transmural injury? ✅st seg elevation

What is subendocardial injury? ✅inner 1/3 to inner 1/2 of ventricle wall - still able to
pump, but not functionally

What happens to st seg w/ subendocardial injury? ✅st seg depression

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