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NURA 221 FINAL EXAM
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NURA 221 FINAL EXAM
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NURA 221 FINAL EXAM 2024-2025Antidysrhythmic Drugs
drugs used to treat abnormal heart rhythms
begins at the SA node,
descends to the AV node,
down to the bundle of HIS,
to both ventricles
What is the cardiac electrical system pathway?
Automaticity, conductivity
Cardiac dysrhythmias occur usually due to a problem with either __ or ___
Automaticity
The ability to generate an electrical impulse without outside stimulus
Decrease, sinus bradycardia, increase, sinus tachycardia
Cardiac dysrhythmias can come from either a ___ in automaticity causing ___ or an
___ in automaticity causing ___
Conductivity
The ability to conduct an impulse through an internal electrical system
Block, delay
Cardiac dysrhythmias can occur from a __ or __ in conductivity
Asystole, v-tach, V-fib
What are life-threatening dysrhythmias?
,New pattern
harmful, life-threatening
pre-life-threatening
future
The use of antidysrhythmics will depend on:
is this a ___ for this patient?
is this dysrhythmia ___ and/or ___?
is this a ___ rhythm?
is this rhythm that may lead to ___ (near or far) problems?
Resolution, cardiac output, HR, BP, capillary refill, normal
The expected outcome of antidysrhytmic therapy includes ___ of the dysrhythmia,
restoration of the __, __, __, and __, and return to __ state of being
Cardiac output
The amount of volume ejected from the LV over one minute
Class 1: sodium channel blockers
class II: beta-blockers
class III: potassium channel blockers
Class IV: calcium channel blockers
unclassified class: others
What are the antidysrhythmic classes?
1A, 1B, and 1C
Class 1 sodium channel blockers are divided into groups.
Long-term
Class 1A Na channel blockers are for ___ use (dysrhythmias that won't get better)
,Stabilizes, sodium, potassium
Class1A ___ the cell membrane by preventing the ready movement of __ and ___
across the cellular barrier
Impulse conduction, repolarization, atria, ventricles, purkinje fibers
Class 1A decreases __ and delays ___ in the __, ___, and ___
Widening, lengthened
Class 1A Na channel blockers cause ___ QRS and ___ PR-interval on the ECG
SVT, v-tach, a-flutter, a-fib
What are class 1A Na channel blockers used for?
Procainamide (Pronestyl)
What is a class 1A Na channel blocker?
Existing conduction delay
myasthenia gravis, weakness
toxic, >25%, abolition
hold, stop, provider
Class 1A Na Channel Blockers nursing management:
should not be utilized with ___ problems
avoid in ___ patients, may increase muscle ___
watch for ___ effects: widening of the QRS ___ or __ of the P wave
__ or ___ administration and inform ___ ASAP if these are noted
Short term
Class 1B Na channel blockers are for ___ use (have to fix the dysrhythmias right
away)
Electrical conduction, automaticity, repolarization
, Class 1B meds decrease ___ and ___. Clas 1B meds increase ___ rate
Ventricles, v-tach, v-fib, PVCs
Class 1B meds main effect is located within the ___. Helps with __, __, and ___
Lidocaine (Xylocaine)
What is the most popular class 1B Na channel blocker?
Cardiac blocks
levels, toxicity
ECG, IV
immediately, prolonged, prolonged, aggravation
Class 1B nursing management:
do not use with severe ___
check blood ___ often to monitor for ___
constant __ monitoring is necessary for __ admin
stop infusion ___ if any toxic signs are noted: __ PR interval, ___ QRS complex, or
___ of existing dysrhythmia
Conduction, atria, ventricles, purkinje, repolarization
Class 1C Na Channel blockers decrease ___ in __, __, and ___. Class 1C meds
delay ventricular ___
SVT (superventricular tachycardia)
Class 1C Na channel blockers treat and prevent ___
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