ANSWER: Ambulatory venous hypertension Failure of venous valves, causing
bidirectional blood flow and increased pressure to the capillaries.
What occurs when there is low flow in blood capillaries to leukocytes? They
become stuck and release proteolytic enzymes and oxygen-free radicals that
damage the capillary basement membrane.
, What happens to plasma proteins with low flow conditions in blood capillaries? -
Answer Plasma proteins seep into the surrounding tissue, causing a fibrin cuff,
edema, and hypoxia.
What causes reddish brown skin linked with oedema/lymphedema? - Answer High
intracapillary pressure strains endothelial cells apart, and erythrocytes depart the
blood capillaries, coloring the skin with hemosiderin deposits.
Does ambulatory venous hypertension generate an increase or decrease in net
filtration? - Answer: Increase
What causes mechanical insufficiency in CVI - ANSWER Fibrin leaking into the
tissue, as well as an inflammatory ANSWER, overall increase in protein and water
load
What is not considered when staging lymphedema? ANSWER: Limb volume.
Mild lymphedema severity volume increase: <20%.
ANSWER: 20-40% volume increase for moderate lymphedema severity.
Severe lymphedema severity increases by >40%.
DVT forms in subfascial veins.
What is the most frequent ulcer, and where do they form? Venous stasis form in the
distal 1/3 of the leg, typically around the malleoli.
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