100% satisfaction guarantee Immediately available after payment Both online and in PDF No strings attached
logo-home
FoEM CBR22- Cardiology Exam $10.59   Add to cart

Exam (elaborations)

FoEM CBR22- Cardiology Exam

 2 views  0 purchase
  • Course
  • Institution

FoEM CBR22- Cardiology Exam What underlying pathologic process distinguishes myocardial infarction from angina/unstable angina? Atherosclerotic plaque rupture → exposed endothelium → clot attaches → reduced blood flow; if cell death occurs (usually due to complete vascular obstruction) then...

[Show more]

Preview 2 out of 13  pages

  • June 12, 2024
  • 13
  • 2023/2024
  • Exam (elaborations)
  • Questions & answers
avatar-seller
FoEM CBR22- Cardiology Exam
What underlying pathologic process distinguishes myocardial infarction from angina/unstable angina?
Atherosclerotic plaque rupture → exposed endothelium → clot attaches → reduced blood flow; if cell
death occurs (usually due to complete vascular obstruction) then positive trop and MI; if no cell death
occurs then negative trop and angina/unstable angina.


What is the difference between transmural and nontransmural infarction?
Transmural: usually STEMI, large vessel affected, benefit from thrombolytics/PCI; Non-Transmural:
usually NSTEMI, smaller subendocardial artery, may benefit from PCI but no thrombolytics.


What defines Unstable Angina?
Stable Angina + pain at rest, new pain, increasing pain severity, hemodynamic changes with pain.


Acute chest pain at night, EKG with STEMI, all symptoms and EKG changes resolve with nitro?
Prinzmetal's Angina (coronary spasm, most do not have CAD). Treat with CCB.


What are early to late EKG changes with ACS?
Hyperacute T's and Giant R (very early and transient), ST Elevations, ST Depression (ischemia or
reciprocal), Q waves (1 square wide, 1/3 height QRS), T wave inversions.


What syndrome associated with Biphasic T-wave in V2/V3?
Wellens Syndrome: biphasic (type A) or deeply inverted, symmetric (type B) T wave in septal leads =
early signal of proximal LAD lesion.


Chest Pain with STE V1-V4 with STD II, III, aVL
Anterior MI 2/2 LAD occlusion, may affect large territory of LV, septum and conduction system (high
grade blocks, wide complex bradycardias), commonly have shock, possible ruptures


Chest Pain with STE I, aVL, V5, V6 with STD V1
Lateral MI 2/2 LAD vs LCx occlusion, may affect LV


Chest Pain with STE II, III, aVF with STD V1-V4
Inferior MI 2/2 occlusion of PDA (RCA > LCx), may affect AV node (usually transient narrow complex
bradycardias), may cause papillary muscle rupture


Chest Pain with STE III > II and V1 > V2
Right Ventricular MI, should get R-sided leads (STE in V4R, V5R), 2/2 proximal RCA lesion, associated
with Inferior MI.


Chest Pain with STD V1-3
Posterior MI, get posterior leads to dx (req only 0.5 mm elevation for STEMI dx), 2/2 occlusion of
Posterior Descending (RCA > L circ)


What meets STEMI criteria for leads V2-V3 versus all other leads?

, V2-V3: ≥2mm in MEN ≥ 40yrs, ≥2.5mm in MEN < 40yrs, or ≥ 1.5mm in WOMEN; All other leads: STE at
the J-point of ≥ 1 mm in two contiguous lead


What distinguishes Type I-Type V MI?
Type I: MI caused by acute atherothrombotic CAD, usually due to plaque rupture or erosion; Type II:
MI 2/2 mismatch of oxygen supply and demand; Type III: typical MI presentation but death before
biomarkers obtained; Type IV: MI 2/2 PCI; Type V: MI 2/2 CABG


How can you detect MI in patients with paced rhythm or old LBBB
Sgarbossa Criteria: a) STE >1mm with concordant (same direction) QRS, b) concordant STD >1mm V1-
V3, c) STE >5mm with discordant (opposite direction) QRS (modified Sgarbossa changes this last rule
to discordant STE >25% preceding S wave)


What is unique about the management of Inferior MIs?
With Inferior MI, always consider RV involvement and get right-sided ECG leads


What is unique about the management MI with right-ventricular involvement?
They are preload dependent and will become very hypotensive with nitroglycerin - avoid this, give IVF
for hypotension


What are potential early complications (<24hr) of MI?
Arrhythmia (most common), shock 2/2 pump failure or valve dysfunction (valve rupture).


What are potential late complications (>24hr) of MI?
Thromboembolism, myocardial rupture, valve rupture, CHF, pericarditis


What syndrome would you consider in someone with pleuritic chest pain 4wks after MI?
Dressler's syndrome: autoimmune pericarditis, typically 2-6wks s/p MI. Tx it with NSAIDs.


What artery typically supplies the SA node and AV node?
SA- RCA 60%, LCx 40%; AV- RCA 90%, LCx 10%; concern for bradycardias if Inferior MI


What is the cause of cardiac Tamponade after MI?
Myocardial wall rupture, give IVF, bimodal distribution first few days and 1-2 weeks, pericardial
tamponade. Dispo to OR.


What ECG finding is classic in Cardiac Tamponade?
Electrical Alternans


What could cause a new murmur and shock after MI?
Papillary muscle rupture leading to mitral regurgitation, Rx- reduce afterload and dispo to OR; same
treatment if septal wall rupture


What potential treatments for AMI have been shown to reduce mortality?
Defibrillation for VF/VT (30% mortality reduction), Aspirin (25% mortality reduction)

The benefits of buying summaries with Stuvia:

Guaranteed quality through customer reviews

Guaranteed quality through customer reviews

Stuvia customers have reviewed more than 700,000 summaries. This how you know that you are buying the best documents.

Quick and easy check-out

Quick and easy check-out

You can quickly pay through credit card or Stuvia-credit for the summaries. There is no membership needed.

Focus on what matters

Focus on what matters

Your fellow students write the study notes themselves, which is why the documents are always reliable and up-to-date. This ensures you quickly get to the core!

Frequently asked questions

What do I get when I buy this document?

You get a PDF, available immediately after your purchase. The purchased document is accessible anytime, anywhere and indefinitely through your profile.

Satisfaction guarantee: how does it work?

Our satisfaction guarantee ensures that you always find a study document that suits you well. You fill out a form, and our customer service team takes care of the rest.

Who am I buying these notes from?

Stuvia is a marketplace, so you are not buying this document from us, but from seller ACADEMICAIDSTORE. Stuvia facilitates payment to the seller.

Will I be stuck with a subscription?

No, you only buy these notes for $10.59. You're not tied to anything after your purchase.

Can Stuvia be trusted?

4.6 stars on Google & Trustpilot (+1000 reviews)

78998 documents were sold in the last 30 days

Founded in 2010, the go-to place to buy study notes for 14 years now

Start selling
$10.59
  • (0)
  Add to cart