NR507 Week 7 Quiz 71 CORRECTLY ANSWERED QUESTIONS LATEST UPDATE
Diabetes Type I Pathophysiology
Diabetes Mellitus type 1 Classic Signs
DM Causes
Diabetes Insipidus Kidney Function
DM chronic complications
Commons signs for DM Type I and Type II
DI caused by dysfunction of:
DI Defined
Pancr...
Ans: Autoimmune-mediated: environmental-genetic factors tiggers cell-mediated
destruction of pancreatic beta cells.
Idiopathic or non-immune: Secondary to other disease like pancreatitis.
Autoantigens bind to beta cells and circulate blood and lymph-->Activation of T helper 1 +
2 lymphocytes-->Macrophages with releases of IL and TNFa, T cytotoxic cells, B
lymphocytes to produce islet cells autoantibodies-->Destruction of beta cells with
decreased insulin secretion.
Ans: Type I:
Autoimmune: Environmental-Genetic predisposition
Idiopathic: secondary to other disease (ex. pancreatitis)
Type II:
Genetic Predisposition
Obesity
BOTH: Lack of endogenous insulin
4). Diabetes insipidus kidney function
Ans: Nephrgenic DI: inadequate response of the renal tubules to Anti Diuretic Hormone
(ADH). Acquired or genetic. Gradual onset.
Urine output for DI: 8-12 L/Day.
5). Dm chronic complications
Ans: Neuropathy
Nephropathy
Retinopathy
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, Macrovascular Disease
Infection
6). Commons signs for dm type i and type ii
Ans: Polyuria, polydipsia, fatigue.
7). Di caused by dysfunction of:
Ans: Pituitary System
8). Di defined
Ans: The inability to concentrate urine and the production of copious amounts of dilute
urine.
9). Pancreatic, insulin secreting cells
Ans: Beta cells; endocrine gland.
10). Dm end result on cellular level
Ans: Cellular starvation d/t lack of glucose in cells-->liver stores of glycogen depleted--
>use of fat and protein-->Ketones are byproduct of fat catabolism-->Diabetic Ketone
Acidosis (DKA) due to ketone build up
OR if less severe: Prevention of lysis of fats-->No ketone formation-->Hyperglycemic
Hyperosmolar Nonketotic Coma (HHNK).
11). Polydipsia
Ans: Excessive thirst.
Elevated blood glucose-->water osmotically attracted from cells into blood-->Intracellular
dehydration-->hypothalmic stim. of thirst.
12). Polyuria
Ans: Excessive urination.
Increased blood glucose=> osmotic diuretic--> amount of glucose filtered by glomerulus
is greater than can be reabsorbed-->glycosuria-->large amounts of water lost in urine.
13). Polyphagia
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