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NU643 Week 4 Quiz 2 – Questions & Complete Answers
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NU643 Week 4 Quiz 2 – Questions & Complete Answers
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NU643 Week 4 Quiz 2 – Questions & Complete AnswersTheories of schizophrenia. ✔️Ans - Elevations of dopamine, epigenetics
(genes being turned on and off due to environment), eNMDA/ glutamate
defective genes inherited- environmental pressures turn on defective genes
Mesocortical dopamine pathway ✔️Ans - Neg symptoms: 5 As alogia,
avolition, asociality, anhendonia, affective blunting, VTA to prefrontal
cortex: cognitive changes, slowing of thought (low levels of dopamine in
mesocortical pathway), drugs target this area to inc dopamine (because
there is less dopamine)
Tuberoinfundibular dopamine pathway ✔️Ans - inc prolactin, due to the
hypothamus, blocks dopamine, which in turn increases prolactin (inverse
relationship with dopamine (amenorrhea, galactorrhea (milk in breast)-
risperidone
Nigrostriatal dopamine pathway ✔️Ans - Movement disorders: tardive
dyskinesia, extrapyramidal symptoms, parkinsons's, slow brady-dyskinesia,
tremors
Mesolimbic dopamine pathway ✔️Ans - positive symptoms:
(hallucinations, rapid speech, delusions) hyperactivity in this pathway. The
mesolimbic pathway, sometimes referred to as the reward pathway, is a
dopaminergic pathway in the brain.
Conventional/typical antipsychotics ✔️Ans - haloperidol and
chlorpromazine (1st generation) tend to block dopamine D2 receptors in
all of the dopaminergic pathways of the brain.
atypical 2nd generations ✔️Ans - Less EPS, less movement disorders,
less side effects; Nonselectivity of receptor sites, serotonin (5HT) blocker
also (stimulates dopamine release further down in the brain), dopamine
blocker (dopamine is not blocked as fully as conventional antipsychotics)
EPS ✔️Ans - Caused by blocking of dopamine D2 receptors (risperidone
inc'd risk of EPS in high doses)
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