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NR 507 Mid Term Study Guide (Set-2)/ NR 507 Week 4 Midterm Study Guides, NR 507: Advanced Pathophysiology, Chamberlain $12.49   Add to cart
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NR 507 Mid Term Study Guide (Set-2)/ NR 507 Week 4 Midterm Study Guides, NR 507: Advanced Pathophysiology, Chamberlain

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NR 507 Mid Term Study Guide (Set-2)/ NR 507 Week 4 Midterm Study Guides, NR 507: Advanced Pathophysiology, Chamberlain

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  • May 10, 2023
  • 13
  • 2022/2023
  • Exam (elaborations)
  • Questions & answers

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  • chamberlain
  • nr 507 mid term study guide set 2 nr 507 week 4 midterm study guides
  • nr 507 advanced pathophysiology

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  • NR 507
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NR 507 Week 4 Midterm Study Guides
NR 507: Advanced Pathophysiology

 Verified And Correct Answers
 Best document for Exam
 Secure Better Grades

, Midterm Study Guide
Pulmonary:
 Review concepts related to anticholinergic drugs and the treatment for
asthma
o (Block acetylcholine binding (primarily in lung) ->
bronchodilation through decreased parasympathetic response
 Tiotropium, Ipratropium
o Short-acting beta-adrenergic agonists (SABA) – activate beta
receptors primarily in the lung -> bronchodilation through
increased sympathetic response
 Albuterol
o Oral corticosteroids – inhibit inflammatory cells and chemical
production -> reduce inflammatory process
 Prednisone, prednisolone, methylprednisolone
o Long acting:
 Inhaled corticosteroids
 Beclomethasone, triamcinolone
 Mast cell stabilizers
 Cromolyn
 Leukotriene modifiers
 Zafirlukast, Montelukast
 Long acting beta2 adrenergic agonists
 Salmeterol
 Methylxanthines
 Theophylline
 Monoclonal antibodies
 Omalizumab
 Bronchitis and associated pathogenesis
o In bronchitis, inspired irritants promote bronchial inflammation,
causing bronchial edema, increases the size and number of
mucous glands and goblet cells in the airway epithelium, smooth
muscle hypertrophy with fibrosis, and narrowing of the airways.
o Hypersecretion of thick, tenacious mucus occurs and cannot be
cleared because of impaired ciliary function. The lung’s defense
mechanisms are therefore compromised, increasing
susceptibility to pulmonary infection, which contributes to airway
injury and ineffective repair.
o Frequent infectious exacerbations from bacterial colonization of
damaged airways are complicated by bronchospasm with
dyspnea and productive cough.
 Chronic bronchitis and related acid/base disturbances, perfusion, blood
flow between the heart and lungs

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