100% satisfaction guarantee Immediately available after payment Both online and in PDF No strings attached
logo-home
Summary Histology Rapid Revision Krok 1 $3.24   Add to cart

Summary

Summary Histology Rapid Revision Krok 1

 14 views  0 purchase
  • Course
  • Institution

In this file you will find a Rapid Revision Summary for the Histology part by Medic Mukul of Krok 1. A part of the summary is based on most frequent asked questions as well as ministery questions.

Preview 3 out of 7  pages

  • May 2, 2023
  • 7
  • 2022/2023
  • Summary
avatar-seller
Histology
Rapid Revision KROK




Minna Mamik
@ Medic Mukul

, induced immune
Drug
hemolytic anomia
flare
(Cytexicsensitivityi
wheal a
a




S
Hypersensitivity -> Y
dypes
response
I
Type I
->
Anaphylatic, Immediate, Reagine
a
Adopy hemolytic disease in newban

by Rh-incompatibility
Cytotoxic) Type II -

Antibody Mediated, Complement
dependent
Immuno Complex
Type III ->
Hypersensitivity Thymus ->
Produce thymasin
Hasall's Capuscles present all).
Type E ->
Delayed a T-cell mediated
hypersensitively (degenerated
will shinkover deme
↳ & inducation
erythrema >Digeoge Syndrome -




thymosin not produced
>Undergo Age involution

of allengen with Absence Di-Geage Syndrome (Chromosome
Type I interaction
-




IgE, absorbed on -
22 defect)
(is-30 min)
mastocytes and basophils membeanes

e.g bronchial astma, after
acine introduction
Red Bone Marrow ->
Produce RBC




Type II
- Fixadion of alien
antigens fixed on the cells
Ezydheopoeitin
N
actson Redbone.

(B -

cell by prefamed andibodies with the subsequent kidney produce
mediated)
activation of complement
system by the cell-bound Aputation -> Removal of agans

Immune complexes ->
e.g bloodteansfusion, Rh phantom pain Amputadion

Type III -> Formation of cell-bound immune complexes causalgic pain -> Gun shot


fixing ondo tissues &
producing their
damage protopathic pain -> Glossitis, inflammation
->
e.g Systemic lupus eythomedus, Glomoulonephitis
(SLE)
Cellular Ganuloma of
Type
immune Combination bact &
-> response, Delayedin maccophage
-




T-cell=> > CM1
Granuloma
type It
hypes.
->

e.g
Lapzosy "/Sandwich
palisade Azragment
->

Mantcuxtes fatB
-




->




↳ Geanuloma cell's

(at
Poikilocytosis -


Change shape of RBC Tuberculosis -


Langerhans all skinbarrie)
dencritic alls

Anisocytosis ->
change Size of RBC
lepzosy - Vizchow
call/cigarete-like cells (LoVe)
Rhinoschecoma -
Mikuliz cell

Gumma Bodies
Immune
agains
-> Central &
Peripheral Syphilis
-




↓ *
(hymosin) thymus Tonsils -



B-lymp
(RBC&WBC) Red Bane Marrow Lymph noce

splee langs -
3
types of Cell

Antigen Indipendent) Antigen dependent) pneumocytes/ Prumocytes/ pneumocytes/
Alvedlan cell Alseolar Cell Alvedan Maczophage
Tonsils -
Only lymphoid organ
with epithelial lining Type I Type I
single

~ Nasopharynged ↳
Tonsils; ↳ Simple difussion produce Surfactant
surface
engulffonign body
Tonsil -numerous
cryptspresent (Gas exchange) to reduce



ETabralin
Tubal Acts
Tension as
phagocytosis
Waldeyer's
Tonsil >
hymphocytes arranged act as precuse prent
lungs from
paired het / as
lymphatic nodules fa type II
collapsing Emphysema
↳ Prevent
tendency to ↳ macrophage comes
paladine
Tonsil Tonsil
recede
Ccollapse)
~gese
& breaks walls,

baggy lungs,
Hal/gastzomucin alvedi damaged
Lymph noce ->>Bean shaped >Follicles in Contex
nodles - proten,producus
Medullary cads present aka-lymphatic
parital/oxyntic cell

2
It
Trabecal > cards in medulla
Rods
(duilight vision) contains maximum


Spleen -> Cential
artery Spleen consist of parts2 Alveolar
dype II ~ Mitochondria

↳> white pulp-ecentrically
tiven-> Central rein
placed acteride

LoVe ↳ Red pulp-made of
sinusoids & cads


Minna Mamik

, o Surfactant
Composition; o
Eyelens from Surface Ectodum * Redina from Estodem


Dipalmitoylphosphatidycholine (Dppc) chaloid ->
fom Mesodem


Sphingolipids S ↳ have blood veins


phospholipids CECell
Gganelles
lecithin Endoplasmic Redicutum

Ca2t SmoodhER
Rough ER
or




Sant Agranular
Glanclar


A
protein
W




B
Ribosomes(granules) pt.
Function

Ribosome absent

Function:
Protein Synthesis lipid Synthesis
hydrophillic
-




hydrophobic Hydrophilic staage of Cant
#ate
water Detofication of Daugs
BK Be careful

Golgi Apparatus
St
Capillaries 1) post - translational modification
Non-continuous/Sinusoidal 2)
Continuous Fenestzated
sarding proteins
↳ NO Gaps, Nopores ↳ small Gaps ↳ Wider Gaps ↳
having (is &tzans end
kidney Red Bone Marrow
Brain
e.g eg he e.g;
lungs
Sheletal M.
I Liver

Spleen
↓RBC
WBC
-Expating & famation function
Smooth M. absorption platelets
ect,

hysosomes -> Suicide Bag/Residual Bodies

St Articles--Resistance Vessel Function:Destruction of unwanted Subst, bact, etc
Veins- Capacitance ↳ (produce hydedytic enzymes)
act as
phagocytosis
Lymphatic Capillaries (NoBasement or thin
-




Macula Densa ~membrane penoxisomes -> Destruction of te radicals by Catalase
↳ H202
Oxidation of of FA
long & very long
->
chain


St Ammonification. NHy is
dangerous in beain, heart. Cytoskeleton ->
Provides stength &steucture to cell
Muscles
bone of cell)
Brain, heart ↳ Microdubules -



Big filaments
Transport of NH, of NH3
Transport ↳ Intermediate filament -> in
most quanities present
by Glutamate
↓Livert by Alanine
↳ Microfilament -
Small filaments
lots of H20 NHs

both
used fa Ureacycle -> in
cytoplasm
&
excretion of ammonia ↓ mitochondria 6 Epidhelium
wied

↓ simple epith. ->
Single layered
Albumin
most in
kidney Compound epith ->
Multilayered
Blood lange
~In quanidy
et
Collagen -> MostAbundant in
protein body .
Simple epithstium
Bone, Skin, Tendon epith
Type I Simple Squamos 3B- A (mnemonic
-> ->




if absent Osteogenesis imperfecta Bowman's capsule
-
, -




Blood vessels
Cardilage
-


Type II -


>Body Cavity
if absent Achondroplasia
>
> -> Alvedi


Type III - Redicular fibers, Blood Vessels, Aata -Simple Cuboidal epith. -> T-ok
Like Cube II
Ellen Danlos
Syndrome -
Thyzoid Follides

I -> Basement
Membrane ·7777, Germinal Epith. of Ovary
Type
-AlportSynchrome -
kidney Tubules


Minna Mamik poT & DCT (
proximal & Distal
convoluted Tubules

The benefits of buying summaries with Stuvia:

Guaranteed quality through customer reviews

Guaranteed quality through customer reviews

Stuvia customers have reviewed more than 700,000 summaries. This how you know that you are buying the best documents.

Quick and easy check-out

Quick and easy check-out

You can quickly pay through credit card or Stuvia-credit for the summaries. There is no membership needed.

Focus on what matters

Focus on what matters

Your fellow students write the study notes themselves, which is why the documents are always reliable and up-to-date. This ensures you quickly get to the core!

Frequently asked questions

What do I get when I buy this document?

You get a PDF, available immediately after your purchase. The purchased document is accessible anytime, anywhere and indefinitely through your profile.

Satisfaction guarantee: how does it work?

Our satisfaction guarantee ensures that you always find a study document that suits you well. You fill out a form, and our customer service team takes care of the rest.

Who am I buying these notes from?

Stuvia is a marketplace, so you are not buying this document from us, but from seller minoo. Stuvia facilitates payment to the seller.

Will I be stuck with a subscription?

No, you only buy these notes for $3.24. You're not tied to anything after your purchase.

Can Stuvia be trusted?

4.6 stars on Google & Trustpilot (+1000 reviews)

80796 documents were sold in the last 30 days

Founded in 2010, the go-to place to buy study notes for 14 years now

Start selling
$3.24
  • (0)
  Add to cart