NURS 623 Worksheet for Exam 1NURS 623 Worksheet for Exam 1
The following list is meant to serve as a study guide and may not be inclusive of every component of the exam. You are responsible for the required readings and the Kaltura presentations.
Be prepared to identify diagnosis (or treatment) ...
nurs 623 worksheet for exam 1 the following list is meant to serve as a study guide and may not be inclusive of every component of the exam you are responsible for the required readings and the kaltu
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NURS 623
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NURS 623 Worksheet for Exam 1
The following list is meant to serve as a study guide and may not be inclusive of every
component of the exam. You are responsible for the required readings and the Kaltura
presentations.
Be prepared to identify diagnosis (or treatment) when given a picture with the description of the
various skin problems.
Working through the questions should help point out areas you need to spend more time
further reviewing. Also, please look over the review guide in the module section.
Parasitic skin problems
1. What is the mode of transmission? Contaminated food, water, soil, blood, and can be
transmitted through sexual contact.
2. What is the clinical presentation for each of the parasitic skin problems? Small insects or
worms that burrow into the skin to live and lay their eggs. SUBJECTIVE: intense itching worse
at night.OBJECTIVE: 1-2 mm red papules intense itching excoriations may be present with
crusting from itching.
3. What are the commonly prescribed medications for the various parasitic skin problems?
Permethrin cream (scabies) Permethrin rinse (lice) Ivermectin, lindane
4. What should you include in the patient education to prevent spreading of the various parasitic
skin problems?
Good hygiene, don’t share combs/hats/ wash bedding in hot water, clothing hot water too.
Fungal skin problems
5. What is the clinical presentation of each of the fungal skin problems? Thrive warm moist
environments, folds of skin. Feet / goin. Scaly rash, discoloration of the skin, red, cracked
peeling and biggest thing ITCHES. OBJECTIVE: bright red rash with macules or satellite
lesions seen on the borders, a cardinal symptom is pruritus’ and sometimes burning.
6. How are fungal infections usually diagnosed? By scrapping off the skin and looking under a
microscope Is there a difference if the fungal infection is resistant to treatment? Many of these
cases are completely resistant to one form of another class of antifungal. So combo of antifungal
treatment is commonly used.
7. What are the commonly prescribed medications end in ZOLE or FINE for the various fungal
skin problems? Clotrimazole (Canesten), Econazole (Ecoza), Ketoconazole (Nizoral), Desenex
Miconazole
,8. What should you include in the patient education regarding the various fungal skin problems?
Wash, dry thoroughly, wear socks, wash shoes
Bacterial skin infections
9. Which bacterial skin infection is considered highly contagious? Impetigo What is the “classic”
presentation?Vesicles moist HONEY-Crusts
10. What is the clinical presentation of each of the bacterial skin infections? Induration and
erythema of the affected area with pain out of proportion to overlying skin changes.
11. What is the management of a minor case of folliculitis (non-pharmacologic)? Antibacterial
soap and warm wather, good hand washing, good hygiene, avoid old razors, gentle cleansing bid
with antibacterial soap
12. What are the commonly prescribed medications for folliculitis? Mupirocin (Bactroba) 2%
ointment or cream tid for 5-14days . Retapamulin (Altabax) bid for 5 days. Clindamycin 1%
solution gel, pledget bid until lesions are clear. EES 2% solution lotion or gel bid
13. What is the difference between a carbuncle and furuncle? FURuncle is BOIL. Carbuncle are
multiple boils. Does the treatment differ? Have to be drained, warm compress help if not I & D,
Can use topical abx, systemic not necessary unless cellulitis, or immunocompromised, elderly
etc.
14. What are the considerations when determining treatment for cellulitis? Severity of the
infection, site of the infection, presence of underlying disease, and virulence of the pathogen. For
example, DM2 high risk, pts on corticosteroids higher risk, previous surgeries or surgical site,
Hands feet and face needs aggressive treatment.
15. What are the pharmacologic recommendations for management of skin and soft tissue
infections in primary care? The following are good choices for cellulitis NOT associated by
human or animal bites: Pen VK, dicloxacillin, clindamycin, or cephalexin for 5 days. If allergic
to PCN, clindamycin, azithromycin, or clarithromycin. Infected with human or animal bite then
augmentin for 2 weeks.
Viral skin infections
16. What is the clinical presentation for the various viral skin infections? Warts: Subjective:
small bump or group of bumps that has been present for several weeks to many months and
sometime for years. Asymptomatic. Objective: small or large fleshy or firm growths or lumps.
Which can be raised fairly flat, single, or multiple, isolated, or clusters together to from
cauliflower-like shape
HSV1 & HSV2 SUBJECTIVE: fever sorethroat hypersalivation ulcers on tongue palate lips,
genital area burning or tiching and discharge muschle aches OBJECTIVE: lesions you need to
examine location, appearance, and distribution, and lymph node involvement.
, 17. What are the non-pharmacologic treatment and pharmacologic treatment for each of the viral
skin infections? Warts and HSV 1 and HSV 2
Warts Non-pharmacologic: Salicylic acid and duct tape / surgery
Pharmacologic: Kertoylytic therapy in the form of salicylic acid plasters (Mediplast) or
solution (DuoPlant, Occlusal is a safe non scarring low cost. Boric acid, tretinoin
HSV1 & HSV 2 none pharm pallative and promote healing, Tylenol, OTC abreva if oral,
Denavir
Pharmantiviral drugs
18. What is the basic patient information for each of the viral infections?lesions eye need to see
opthalm, lip lesions no follow up, genetial safe sex practices
Dermatitis
19. Which dermatitis is an inherited skin reaction that begins in infancy?ATOPIC DERMATITS
(ECZEMA) in its early presentation erythematous in appearance with PAPULOVESICULAR
lesions that may ooze and crust. Later stages the rash becomes a red purple color dries and
develops scaling and LICHENFICATION which is exacerbated by scratching resulting from its
highly PRURITIC nature.
20. What is the “atopic triad?” a personal or part of family have all or part of the 3:
1)ASTHMA,
2) ALLERGIC RHINITIS,
3) Eczema
21. What dermatitis is associated with the expression “the itch that rashes?”Eczema
22. What is the objective finding with atopic dermatitis? Usually begins as infantile eczema with
lesions affecting the cheeks, face, and upper extremities. Erythrema is often seen before
pruritius and the acute lesions are excoriated, maculopapular, and inflamed. In infance and early
childhood, oozing and crusting erythema. Adults: symmetrical lesions that are crusting and
excoriated. Eruthemtous, papulovesicular edematous and weeping. Later rash becomes crusted
scally thickened and LICHENIFIED.
23. What is the primary aim in management of atopic dermatitis? Control
24. What is the non-pharmacologic and pharmacologic management for atopic dermatitis? The
management of dermatitis embodies the fundamental principles of dermatology preciptatin
factors should be eliminated, wet lesions should be dried, dried lesions should be dehydrated,
and inflammation should be treated with corticosteroids.
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