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PEDIATRIC NURSING ALL REVISION QUESTIONS AND CORRECT ANSWERS (GRADED 100%) (2024 UPDATE)
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Sickledex is easy: fingerstick and yields accurate results in 3 minutes. But electrophoresis is needed to distinguish between children with the trait and those with the disease - ANSWERwhat tests are done for Sickle? Preventing exposure to infection, maintaining hydration, and treating oxygen and...

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  • October 17, 2024
  • 84
  • 2024/2025
  • Exam (elaborations)
  • Questions & answers

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PEDIATRIC NURSING ALL REVISION QUESTIONS

AND CORRECT ANSWERS (GRADED 100%) (2024

UPDATE)

Sickledex is easy: fingerstick and yields accurate results in 3 minutes. But electrophoresis is

needed to distinguish between children with the trait and those with the disease - ANSWER-

what tests are done for Sickle?

Preventing exposure to infection, maintaining hydration, and treating oxygen and bed rest -

ANSWER- what is the focus of treatment for sickle cell?


Pain - ANSWER- without adequate hydration in sickle cell, ___ will not be controlled


30 - ANSWER- elevate the HOB no more than ___ degrees in sickle cell


High protein, calorie, and folic acid supplements - ANSWER- diet for sickle cell.


Vaso-occlusive crisis of sickle: caused by stasis of blood - ANSWER- manifestations of this:

fever, painful swelling of hands, feet, and joints, abdominal pain.

Splenic sequestration: caused by pooling and clumping of blood in the spleen (hypersplenism) -

ANSWER- manifestations: profound anemia, hypovolemia, and shock.


Hyperhemolytic cries: accelerated rate RBC destruction - ANSWER- manifestations: anemia,

jaundice, reticulocytosis.

,Aplastic crisis: caused by diminished production and increased destruction of rbcs, triggered by

viral infection or depletion of folic acid - ANSWER- manifestations: profound anemia and

pallor.


YES! Incredibly: pneumococcal, meningococcal, and annual influenza vaccine - ANSWER- is

it important for a child with sickle to receive vaccinations?


Administration is avoided because of the risk of normeperidine- induced seizures - ANSWER-

why is the administration of meperidine (Demerol) avoided in sickle cell?


Microcytic/hypochromic - ANSWER- iron deficiency anemia is a ___ and __ anemia


Z track - ANSWER- what type of injection for iron administration?


Determined by bone marrow aspiration (shows conversion of red bone marrow to fatty bone

marrow) - ANSWER- what is definitive diagnosis of aplastic anemia?


Focuses on restoring function to the bone marrow and involves immunosuppressive therapy and

bone marrow transplantation (treatment of choice if a suitable donor exists) - ANSWER-

treatment for aplastic anemia.


Aplastic anemia - ANSWER- anti-lymphocyte globulin or anti-thymocyte globulin may be

prescribed to suppress autoimmune response in what; colony stimulating factors may be prescribed

to enhance bone marrow production; corticosteroids adn cyclosporine (sandimmune) may be

prescribed, may need blood transfusions.


Hemophilia A or classic hemophilia - ANSWER- factor VIII definiciency may also be known

as what.

,Hemophilia B or Christmas - ANSWER- factor IX deficiency may also be known as what.


X- males are affected most - ANSWER- hemophilia is an __ linked recessive genetic disorder.

Who are affected most?


DDAVP (synthetic form of vasopressin) - ANSWER- ___, a synthetic form of vasopressin,

increases plasma factor VIII and may be prescribed to treat mild hemophilia.


Von Willebrand disease - ANSWER- this disorder causes platelets to adhere to damaged

endothelium; the ___ _____ factor protein also serves as a carrier protein for factor VIII.


Von Willebrand disease - ANSWER- this is characterized by an increased tendency to bleed

from mucous membranes.


Thalassemia major - ANSWER- this type of thalassemia results in severe anemia that requires

transfusion to sustain life (Cooley's anemia).


Thalassemia intermedia - ANSWER- This is manifested as splenomegaly and moderate to

severe anemia.


Thalassemia trait - ANSWER- This produces mild microcytic anemia of thalassemia.


Thalassemia minor - ANSWER- This is an asymptomatic silent carrier case of thalassemia.


B thalassemia major - ANSWER- this is characterized by an autosomal recessive disorder

characterized by the reduced production of one of the globin chains in the synthesis of hemoglobin

(both parents must be carriers to produce a child with beta thalassemia major).

, Supportive; the goal of therapy is to maintain normal hemoglobin levels by the administration of

blood transfusions, may need bone marrow transplantation and splenectomy - ANSWER- what

is treatment for Beta thalassemia major?


B thalassemia major - ANSWER- s/sx: frontal bossing, maxillary prominence, wide set eyes

with a flattened nose, greenish yellow skin tone, hepatosplenomegaly, severe anemia, and

microcytic/hypochromic red blood cells.

With chelation therapy: Exjade or deferoxamine (Desferal) may be prescribed to treat it and

prevent organ damage from the elevated levels of iron caused by multiple transfusion therapy. -

ANSWER- how is iron overload treated in B thalassemia major?


Acute lymphocytic leukemia - ANSWER- ____ _____ ______ is the most frequent type of

cancer in children.


Induction - ANSWER- this phase of chemotherapy achieves a complete remission or

disappearance of leukemic cells.


Intensification or consolidation therapy - ANSWER- this phase of chemotherapy decreases the

tumor burden further.


Central nervous system prophylactic therapy - ANSWER- this phase of chemotherapy prevents

leukemic cells from invading the central nervous system.


Maintenance - ANSWER- this phase of chemotherapy serves to maintain the remission phase.


Leukemic blast (immature) phase cells - ANSWER- a positive bone marrow biopsy would

show what.

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