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Exam (elaborations)

CCRN AACN REVISION QUESTIONS WITH CORRECT ANSWERS

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CCRN AACN REVISION QUESTIONS WITH CORRECT ANSWERS

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  • October 1, 2024
  • 55
  • 2024/2025
  • Exam (elaborations)
  • Questions & answers
  • AACN
  • AACN
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CCRN AACN REVISION QUESTIONS
WITH CORRECT ANSWERS



atrial fibrillation adverse consequences - Answer - 1. decreased cardiac output due to loss of atrial kick,
rapid ventricular rate, irregular ventricular rhythm



2. tachycardia induced cardiomyopathy - in rapid afib for prolonged period of time



3. thromboembolism



right bundle branch - Answer - -right side of the interventricular septum and right ventricle

-impulse travels slower as the right ventricle is smaller/thinner



left bundle branch - Answer - two main divisions: anterior fascicle and posterior fascicle carrying
impulses to the left ventricle



PR interval - Answer - delay of AV node to allow filling of ventricles



QRS complex - Answer - ventricular depolarization



shape depends on the lead that is being monitored and the ventricular activation device



T wave - Answer - ventricular repolarization



normally in the same direction as the QRS

,upright, flat, inverted



pathologies of T wave - Answer - MI, E/L levels, drug effect, myocardial disease, and lead being
recorded



u wave - Answer - repolarization of the purkinje fibers



SHOULD BE POSITIVE especially when T wave is positive



large u waves can be seen when repolarization is abnormally prolonged - E/L imbalances like
hypokalemia, hypocalcemia, hypomagnesemia, IICP, LVH, certain medications



ST segment - Answer - early ventricular repolarization



should be at isoelectric line



J point - Answer - where QRS complex ends and ST segment begins



QT interval - Answer - ventricular depolarization and repolarization varies with age, gender, and heart
rate



beginning of the QRS to the end of the T wave



QT must be corrected to a HR of 60 bpm



QTc - Answer - corrected QT interval =

QT/(square root of R-R interval)



normalizes for HR

,long QTc --> torsades, ventricular arrhythmia, Vfib



vertical axis - Answer - each small box is 1mm or 0.1 mV



each large box is 5mm or 0.5 mV



most common complication of ischemic heart disease and MI - Answer - dysrhythmias



best leads for differentiating wide QRS rhythms - Answer - v1 and v6



v1 and v6 - Answer - helps to differentiate VTACH from SVT with aberrant intraventricular conduction



helps to recognize right and left bundle branch blocks



differentiates between right and left ventricular ectopy



differentiates between right and left ventricular pacing



v1 and v6 placement - Answer - v1 - fourth intercostal space at the right sternal border



v6 - left midaxillary line at the v4 level (fifth intercostal space midclavicular line)



primary dysrhythmia monitoring lead - Answer - V1



what is lead II used for? - Answer - used to identify atrial activity if unclear in other leads or for
visualization of R waves during synchronized cardioversion



rhythms with a short PR interval - Answer - may indicate presence of accessory pathway

, lead III or avF - Answer - assists in diagnosis of hemiblock



allows identification of retrograde P waves



allows identification of atrial flutter waves



ectopic beat - Answer - Cardiac beat starting at a point other than the SA node



could be from atria, AV junction, ventricles



sinus bradycardia causes - Answer - inferior MI, OSA, IICP, CNS conditions like stroke, hypothyroidism,
hypothermia, some infectious diseases



digitalis, beta blocker, CCB, ivabradine, antiarrhythmics



Ivabradine - Answer - Cardio

MOA: selective inhibition of funny sodium channels, prolonging slow depolarization phase (phase 4);
decreases SA node firing; negative chronotropic effect without inotropy; reduces cardiac O2
requirements

Use: chronic stable angina in patients who cannot take beta blockers; chronic HF with reduced ejection
fraction

Tox: luminous phenomena/visual brightness, hypertension, bradycardia



medications that cause ST - Answer - atropine, isoproterenol, epinephrine, dopamine, dobutamine,
levo, nitroprusside



persistent tachycardia - Answer - decreased stroke volume, decreased cardiac output, and decreased
coronary artery perfusion secondary to decreased diastolic time that occurs with rapid heart beats



sinus dysrhythmia - Answer - sinus node discharges irregularly and commonly associated with phases of
respiration

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