24 year old patient come in with purulent arthritis of the knee, oligoarticular joint pain, and few vesicle/pustular lesions on her extremities. What is it? - ANSWERS-neisseria GONORRHEA
can cause polyarthralgia, dermatitis, and tenosynovitis
especially in young sexually active ppl
septi...
3RD UWORLD SET QUESTIONS AND ANSWERS 100% 24 year old patient come in with purulent arthritis of the knee, oligoarticular joint pain, and few vesicle/pustular lesions on her extremities. What is it? - ANSWERS -neisseria GONORRHEA can cause polyarthralgia, dermatitis, and tenosynovitis especially in young sexually active ppl septic arthritis common with this in young sexually active adults .ABG on advanced CKD? - ANSWERS -advanced is like creatinine above 3 ANION gap metabolic a cidosis .Acute tubular necrosis with vacuolar degeneration and ballooning of proximal tubular cells? Oxalate crystals are seen. - ANSWERS -ethylene glycol intoxication .All patients with etanercept need to be tested for what? - ANSWERS -TB cuz its a decoy TNFa receptor thing -> can sequester TB in macrophages if its there .Anatomical landmark for doing lumbar puncture? - ANSWERS -iliac CREST -> highest points of iliac crest are at L4 vertebral body .CH50 and AH50 in MAC deficiency? - ANSWERS -LOW for both CH -> this is for CLASSICAL (even if it says complete) cuz both lead to MAC .Complications of EPO -stimulating agents? - ANSWERS -patients can develop HYPERTENSION and increased thromboembolic events or CLOTS .Describe CF diagnosis stuff? - ANSWERS -more sodium REABSORPTION for nasal transepithelial potential difference -> leads to CF diagnosis (more negative) nasal transepithelial potential difference -> cl cant go out (cuz CFTR is malfolded), so sodium (with water) come BACK INTO THE EPITHELIUM (this is abnormal, seen in CF) this leaves the lumen more NEGATIVE in skin -> CFTR is reversed. It absorbs Cl. So can't absorb, NaCl builds up and can be detected on sweat .Dude has hypoglycemia, but no symptoms of hypoglycemia except hes hungry. What happened? - ANSWERS -a non -selective Beta blocker like propanolol was used remember, in low glucose -> NE/E is ramped up (this is what causes tachy, tremors, anxiety/aroual) and B -blockers will block this still hungr y due to ACh effects -> ACh causes the sweating, hunger, and paresthesias (due to muscle activate) .Early after an MI, where does the pericarditis occur? - ANSWERS -its called peri -infarction pericarditis -> 2-4 days after just develops overlying the necro tic myocardial segment dressler's is diffuse autoimmune attack of the entire pericardium (weeks to months after an MI) .Histology of HSP? - ANSWERS -perivascular neutrophilic inflammation small vessels with FIBRINOID NECROSIS leukocytoclastic vasculitis (nuclear debris) this disease presents with palpable purpura, ARTHRALGIAS (JOINT PAIN), and KIDNEY involvement .How can rhabdo effect the kidney? - ANSWERS -causes ATN!!!!! the heme pigement injuries the kidneys -> ATN .How do class 1a antiaryhtmics work and what are they? - ANSWERS -block sodium channel (some can block K too -> prolong AP) these are procainamaide, quinidine .How do class 1b antiarryhtmics work and what are they? - ANSWERS -LIDOCAINE block sodium channe l .How do class 1c antiarrythmics work and what are they? - ANSWERS -block sodium channel flecainide
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