In this file you will find a Rapid Revision Summary by Medic Mukul for the Pathomorphology (pathological anatomy) part of Krok 1. A part of the summary is based on most frequent asked questions as well as ministery questions.
process that
parents & Stops Bleeding
before it spreads Keeps blood within
damaged blood vessel
&
prostate undergoes hyperplasia
hyperd leophy
Pamaryhemostasstriction of Damaged
- leads to Benign
prostatic Hyperplasia eseen
*
H ↓
Carcinogenic agent -
cause cance
plateletAdhesion to discupted Vessel Surface
Japanese
Asbestos -
lung Carcinomas Mesodhelioma
(pork)
platelet degranulation
I ->
Edema-
L↑ vasculan
platelet Aggregation
Food* Nitrosamines permeabil
In
- stomach Carcinoma - Found in smoked o
Fermented Food case of type
polycydic Azomatic Compounds -
Highly carcinogenic
C fibainogen -
usedas
linking molecule
hypersensitivity
Fa fatming of platelet
Plug (weak)
of
the amount
Benzeen
Zings
, Minna Mamik
Problems in
primary Hemostasis can cause >Sepsis (E.coli &N. Meningitis)
Disanders; Adenocarcinoma -> activates
Muain Coagulation
Immune
Thrombocytic Acute leukemia
purpura ->
Promycolytic ->
primary granules
Autoimmune production of IgG against plateletAntigen M coagulation
activate
↳ Most Common cause oftrombocytopenia kids & in
↳ lab shows:↓ Fibzinogen, platelet I
Adults.
↳ Elevated D-Dimen Best Saning test fa DIC
Secondary hemostasis -
stabilize the weak platelet plug via
Coagulation Thrombosis Pathological formation of
=
an Intravascular
Thrombin leads Cascade
Theombin blood clot (thrombin)
to I
generate ->
prevents
hemouchage 1,Fibzin hemachage ↳
Blood ↓
Fibzinogen Can occur in
artery a Vein
↓
Protein Goss-link ↳ M.C. location is the Deep vein (DV) of leg below kre
↓
liver / Cirhosis
I
dot
formation
yield stable platelet Fatty
Fibrin Thombus
Gross, Pathological Feature of Alcoholic liver - Soft, yellow &
Hemanthrosis Deep dissue & liver Geasy
->
bluding into muscle joint Steatosis-Faddy changes CRevensible in
stage)
Hemophilia A -> Genetic facta * deficiency
4 x-unked
-> of
RB Recessive (affects boys most) Degeneration -
(Q)
DG Q found
Fatty/Adipose Degeneration
-
-
droplets
Fat in
Hemophilia B -Genetic factor IX
deficiency (akagiseistmas Myocardial Sancoplasm
->
With Sudan-/ Black(stain)
morie
gives yellow Red colour
Haal
-
facta XI" deficiency
-
Hemophilia C->Genetic
Hyaline
Degeneration Homogenous Glassy
en
-
appearance -
Kyai
↳
May Occue in Fibzous tissue in scous
Von Willebrand Disease (VWF) -
blood doesn't
dot
properly & Some tumos due to deposition of
↳ Genetic VII
deficiency (v.W Factal Glycopzoteins
↳ Disade (a) Zenken's
Mostcommon inherited Coagulation degeneration ->
Glassy a Wax
Treadment;Desmopiessin (ADH -
↳ Minodhnombi
Widespread -> Result
in Ischemia Calcium Acc->
&Infarction
>Dystrophic Calcification -> Dead dissue
↳ >Metastatic Calcification
causes Tissue
=> Thomboplastin, Snake Bite, ->
living dissue
Dagnancy/Abadion
↳ von Koss*
(specialslain)
can also lead to Septicemia, -> cart appears black
blood
poising by bacteria
↳ than Septic Shock
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