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NUR 376 Pathophysiology Exam 1 Questions With Complete Solutions.
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NUR 376 Pathophysiology Exam 1 Questions With Complete Solutions.
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NUR 376 Pathophysiology Exam 1 Questions With Complete Solutions Phases Lof LInflammation: Vascular LPermeability L- L(correct Lanswer) L-Inflammation Lstimulates Lblood Lvessel Ldilation Land Lopening Lof Lcapillary Lpores. LCapillary Lpores Lallows Lfluid Land Lcells L(WBCs Land Lplatelets) Lto Larrive Lat Lsite Lof Linjury. Phases Lof LInflammation: Cellular LChemotaxis L- L(correct Lanswer) L-Chemical Lsignal Lthat Lcalls Lout Lto LWBC's Lto Lsite Lof Linjury. LResults Lin Lleukocytosis, Lwhere LWBCs Lare Lreleased Lfrom Lthe Lbone Lmarrow Land Linto Lthe Lblood Lstream. L **Doctor Lis Lable Lto Lanalyze Lthe Ltype Lof LWBC Land Lnumber Lof LWBCs Lto Ldetermine Ltype Land Lseverity Lof Linfection. Phases Lof LInflammation: Systemic LResponses L- L(correct Lanswer) L-Patient Lexperiences Lsymptoms Lsuch Las: -Fever -lymphadenopathy L(swollen Llymph Lnodes) -pain -sleepiness -lethargy -anemi a -weight Lloss ** LInflammatory Lmediators Lsuch Las Lprostaglandins, LTNF-alpha, Land LILs Lare Lresponsible Lfor Lmany Lof Lthese Leffects Mediators Lof LInflammation L(5) L- L(correct Lanswer) L-1. LCytokines L(Interleukins LILs L& LTNF-alpha) L-either Lamplify Lor Ldeactivate Linflammatory Lresponse -stimulate Lliver Lto Lrelease Lacute Lphase Lproteins 2. LChemokines - Lproteins Lthat Lattract LWBCs Lto Lsite Lof Linjury L 3. LAcute Lphase Lproteins -Facilitates LWBC Lphagocytosis L -Include: LC Lreactive Lprotein L(CRP), Lfibrinogen, Lserum Lamyloid LA, Land Lhecidin 4. LProstaglandins 5. LPyrogens What Lis Lacute Linflammation? L- L(correct Lanswer) L-Short Lterm Linflammatory Lresponse Lthat Lresolves Lonce Linfection Lhas Lbeen Lresolved. What Lis Lchronic Linflammation? L- L(correct Lanswer) L-Ongoing Linfection Lfor La Llong Lperiod Lof Ltime L Ex: LTB Land Lautoimmune Ldiseases Stages Lof LInfection L(5) L- L(correct Lanswer) L-1. LIncubation LPeriod L(no Lidentifiable Lsymptoms) 2. LProdromal LStage L(initial Lappearanc e Lof Lsymptoms - LMOST LCONTAGIOUS 3. LAcute Lstage L(full Linfectious Lexperience) 4. LConvalescent L(body Lfighting Lback, Lstart Lto Lfeel Lrelief) 5. LResolution L(Total Lelimination Lof Linfection) Immunocompetence Lvs. LImmunosuppression L- L(correct Lanswer) L-Immunocompetenc e Lrefers Lto Lthe Lindividuals Lability Lto Lprotect Loneself Lfrom Linfectious Lagents Lbecause Lof La Lstrong Limmune Lsystem. LImmunosuppression Lindicates La Ldefective Limmune Lsystem Lthat Lplaces Lperson Lat Lhigher Lrisk Lfor Linfection. Portals Lof LEntry L(4) L- L(correct Lanswer) L-1. LSkin 2. LRespiratory 3. LGI Ltract 4. LUrogenital Ltracts Atrophy L- L(correct Lanswer) L-"Shrinking" When Lcells Lcant Lmeet Lmetabolic Lrequirements, Lthey Lshrink Lto Ldecrease Lthe Ldemand Land Lincrease Lefficiency. Metaplasia L- L(correct Lanswer) L--Replacement Lof Lone Lcell Lby Lanother Lcell Ltype L -Generally Loccurs Lin Lresponse Lto Lchronic Linflammation to Lenable Ltissue Lsurvival Role Lof Lpyrogens Lin Lfever L- L(correct Lanswer) L-Activate LProstaglandins Lto Lreset Lthe Lhypothalamic Ltemperature -regulating Lcenter Lto La Lhigher Llevel Apoptosis L- L(correct Lanswer) L-Genetically Lprogrammed Ldeath Lon Lcells Lthat Lhas Lno Ladverse Leffects Lon Lbody Antibody Ltiter L- L(correct Lanswer) L-Level Lof Lantibody Lin Lthe Lbloodstream Land Lcorresponds Lto Lthe Llevel Lof Lexposure Lto Lthe Lmicrobe What Lis Lthe Lfirst LIg L(immunoglobin) Lto Lrise Lduring Linfection? L- L(correct Lanswer) L-
IgM Innate Limmunity Lis? L- L(correct Lanswer) L-1st Lline Lof Ldefense Lagainst Linfection Nonspecific Lmechanism Lthat Ldefends Lthe Lbody Limmediately Lagainst Lall Ltypes Lof Lpathogens Passive -acquired Ladaptive Limmunity L- L(correct Lanswer) L--Individual Lgiven Lpre Lmade, Lfully Lformed Lantibodies Lagainst Lan Lantigen -Provides Limmediate Lbut Lshort Lterm Limmunity. LEx: LInfant Lacquiring Lantibodies Lin Lbreast Lmilk Active Lacquired Ladaptive Limmunity L- L(correct Lanswer) L-Longer Llasting Limmunity Lthan Lpassive -acquired Ladaptive Limmunity Lbut Lnot Lpermanent. Ex: LVaccine Adaptive Limmunity Lis? L- L(correct Lanswer) L--2nd Lline Lof Ldefense Lagainst Linfection -Developed Lwith Lexposure Lto Lantigens Land Ltargets Lparticular Lpathogens -Includes LB Land LT Lcells Lto Lfight Linfection Land Lcreate Lantibodies Two Lmajor Lcategories Lof Ladaptive Limmunity L- L(correct Lanswer) L-1. LB Llymphocyte Limmunity L(humoral) 2. LT Llymphocyte Limmunity L(cell-mediated) Aortic LInsufficiency L- L(correct Lanswer) L-Occurs Lduring LS2, Lwhen Lthe Lvalve LSHOULD Lclose Lbut Ldoesn't - LCauses Lblood Lleaks Lback Linto Lthe Lthe Lleft Lventricle -Forward Lflow Lis Lreduced What Lhappens Lto Lthe Lheart Ldue Lto Lmitral Lvalve Linsufficiency? L- L(correct Lanswer) L-
-Aka Lmitral Lregurgitation -Valve Ldoes Lnot Lclose Lproperly -Caused Lby LMI -When Lventricle Lcontracts, Lblood Lleaks Linto Latrium Lcausing Latrial Lenlargement Land Ldecreased Lforward Lflow - LMay Llead Lto LA-fib Land Lvein Ldistention L -Increased Lrisk Lfor LHF What Lcauses LAortic LStenosis? L- L(correct Lanswer) L-Calcific Laortic Lscleros is, Lwhich Loccurs Lbecause Lof Latherosclerosis Land Laging, Lcauses Lstenotic Ldeformity Lof Lthe Laortic Lvalve
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